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@ARTICLE{Tigu:306669,
author = {A.-B. Tigu and M. Nistor and D. Gulei and C.-S.
Constantinescu and D. Kegyes and D. Cenariu and X. Muresan
and R. Munteanu and R. Feder and C. Jitaru and A. Bancos and
M. Santa and R. Tomai and M. Damian and A. Ivancuta and I.
Rus and A. Bojan and M. Zdrenghea and A.-D. Buzoianu and A.
Tanase and H. Einsele and S. Kobold$^*$ and C. Tomuleasa},
title = {{CAR}ing about autoimmune disorders. {U}se of chimeric
antigen receptor engineered {T}-cells in autoimmune
diseases.},
journal = {Blood reviews},
volume = {nn},
issn = {0268-960X},
address = {Burlington, Mass.},
publisher = {Harcourt},
reportid = {DKFZ-2025-02662},
pages = {nn},
year = {2025},
note = {epub},
abstract = {Chimeric Antigen Receptor (CAR) T cell therapy, initially
developed for hematologic malignancies, has recently emerged
as a promising modality for treating autoimmune diseases.
This review explores the evolving role of CAR T cells in
reprogramming immune tolerance and achieving durable
remission in autoimmune disorders. By engineering T cells to
target pathogenic B cells or autoreactive T cells, CAR T
therapy offers a targeted and potentially curative approach
for diseases such as systemic lupus erythematosus, multiple
sclerosis, and type 1 diabetes. Early clinical trials and
preclinical models have demonstrated the feasibility,
safety, and efficacy of CD19-targeted CAR T cells in
depleting autoreactive B cells and restoring immune
homeostasis. Furthermore, next-generation CAR
designs-including regulatory T cell-based CARs and
antigen-specific constructs-highlight the growing precision
and versatility of this platform. Despite these advances,
challenges remain, including potential toxicity, antigen
escape, and the need for long-term immune monitoring. This
review summarizes current findings, delineates mechanistic
insights, and discusses future directions for optimizing CAR
T cell therapies in the context of autoimmunity.},
subtyp = {Review Article},
keywords = {Autoimmune diseases (Other) / CAR T cells (Other) /
Emerging therapies (Other)},
cin = {MU01},
ddc = {610},
cid = {I:(DE-He78)MU01-20160331},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:41318274},
doi = {10.1016/j.blre.2025.101354},
url = {https://inrepo02.dkfz.de/record/306669},
}