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B180
Proteostase neurodegenerativer ErkrankungenID | I:(DE-He78)B180-20160331 |
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Journal Article
Drosophila appear resistant to trans-synaptic tau propagation.
Brain communications 6(4), fcae256 (2024) [10.1093/braincomms/fcae256]
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Identification of cis-acting determinants mediating the unconventional secretion of tau.
Scientific reports 11(1), 12946 (2021) [10.1038/s41598-021-92433-3]
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Autophagy inhibition rescues structural and functional defects caused by the loss of mitochondrial chaperone Hsc70-5 in Drosophila.
Autophagy 17(10), 3160-3174 (2021) [10.1080/15548627.2020.1871211]
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Changes in Glutathione Redox Potential Are Linked to Aβ42-Induced Neurotoxicity.
Cell reports 24(7), 1696 - 1703 (2018) [10.1016/j.celrep.2018.07.052]
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Unconventional Secretion Mediates the Trans-cellular Spreading of Tau.
Cell reports 23(7), 2039 - 2055 (2018) [10.1016/j.celrep.2018.04.056]
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Bin1 directly remodels actin dynamics through its BAR domain.
EMBO reports 18(11), 2051 - 2066 (2017) [10.15252/embr.201744137]
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The Drosophila KIF1A Homolog unc-104 Is Important for Site-Specific Synapse Maturation.
Frontiers in cellular neuroscience 10, 207 (2016) [10.3389/fncel.2016.00207]
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Cellular and molecular modifier pathways in tauopathies: the big picture from screening invertebrate models.
Journal of neurochemistry 137(1), 12 - 25 (2016) [10.1111/jnc.13532]
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Impaired retrograde transport by the Dynein/Dynactin complex contributes to Tau-induced toxicity.
Human molecular genetics 24(13), 3623 - 3637 (2015) [10.1093/hmg/ddv107]
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