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@ARTICLE{Pajtler:119351,
author = {K. Pajtler$^*$ and S. C. Mack and V. Ramaswamy and C. A.
Smith and H. Witt$^*$ and A. Smith and J. R. Hansford and K.
von Hoff and K. D. Wright and E. Hwang and D. Frappaz and Y.
Kanemura and M. Massimino and C. Faure-Conter and P. Modena
and U. Tabori and K. E. Warren and E. C. Holland and K.
Ichimura and F. Giangaspero and D. Castel and A. von
Deimling$^*$ and M. Kool$^*$ and P. B. Dirks and R. G.
Grundy and N. K. Foreman and A. Gajjar and A. Korshunov$^*$
and J. Finlay and R. J. Gilbertson and D. W. Ellison and K.
D. Aldape and T. E. Merchant and E. Bouffet and S.
Pfister$^*$ and M. D. Taylor},
title = {{T}he current consensus on the clinical management of
intracranial ependymoma and its distinct molecular
variants.},
journal = {Acta neuropathologica},
volume = {133},
number = {1},
issn = {1432-0533},
address = {Berlin},
publisher = {Springer},
reportid = {DKFZ-2017-00106},
pages = {5 - 12},
year = {2017},
note = {Consensus Paper},
abstract = {Multiple independent genomic profiling efforts have
recently identified clinically and molecularly distinct
subgroups of ependymoma arising from all three anatomic
compartments of the central nervous system (supratentorial
brain, posterior fossa, and spinal cord). These advances
motivated a consensus meeting to discuss: (1) the utility of
current histologic grading criteria, (2) the integration of
molecular-based stratification schemes in future clinical
trials for patients with ependymoma and (3) current therapy
in the context of molecular subgroups. Discussion at the
meeting generated a series of consensus statements and
recommendations from the attendees, which comment on the
prognostic evaluation and treatment decisions of patients
with intracranial ependymoma (WHO Grade II/III) based on the
knowledge of its molecular subgroups. The major consensus
among attendees was reached that treatment decisions for
ependymoma (outside of clinical trials) should not be based
on grading (II vs III). Supratentorial and posterior fossa
ependymomas are distinct diseases, although the impact on
therapy is still evolving. Molecular subgrouping should be
part of all clinical trials henceforth.},
cin = {B062 / L101 / G380},
ddc = {610},
cid = {I:(DE-He78)B062-20160331 / I:(DE-He78)L101-20160331 /
I:(DE-He78)G380-20160331},
pnm = {312 - Functional and structural genomics (POF3-312)},
pid = {G:(DE-HGF)POF3-312},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:27858204},
pmc = {pmc:PMC5209402},
doi = {10.1007/s00401-016-1643-0},
url = {https://inrepo02.dkfz.de/record/119351},
}