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@ARTICLE{Cosentino:119769,
      author       = {K. Cosentino$^*$ and A. J. García-Sáez$^*$},
      title        = {{M}itochondrial alterations in apoptosis.},
      journal      = {Chemistry and physics of lipids},
      volume       = {181},
      issn         = {0009-3084},
      address      = {Amsterdam [u.a.]},
      publisher    = {Elsevier Science},
      reportid     = {DKFZ-2017-00396},
      pages        = {62 - 75},
      year         = {2014},
      abstract     = {Besides their conventional role as energy suppliers for the
                      cell, mitochondria in vertebrates are active regulators of
                      apoptosis. They release apoptotic factors from the
                      intermembrane space into the cytosol through a mechanism
                      that involves the Bcl-2 protein family, mediating
                      permeabilization of the outer mitochondrial membrane.
                      Associated with this event, a number of additional changes
                      affect mitochondria during apoptosis. They include loss of
                      important mitochondrial functions, such as the ability to
                      maintain calcium homeostasis and to generate ATP, as well as
                      mitochondrial fragmentation and cristae remodeling.
                      Moreover, the lipidic component of mitochondrial membranes
                      undergoes important alterations in composition and
                      distribution, which have turned out to be relevant
                      regulatory events for the proteins involved in apoptotic
                      mitochondrial damage.},
      subtyp        = {Review Article},
      keywords     = {Cytochromes c (NLM Chemicals)},
      cin          = {B160},
      ddc          = {540},
      cid          = {I:(DE-He78)B160-20160331},
      pnm          = {312 - Functional and structural genomics (POF3-312)},
      pid          = {G:(DE-HGF)POF3-312},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:24732580},
      doi          = {10.1016/j.chemphyslip.2014.04.001},
      url          = {https://inrepo02.dkfz.de/record/119769},
}