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@ARTICLE{Beisel:120669,
author = {C. Beisel$^*$ and A. Benner$^*$ and C. Kunz$^*$ and A.
Kopp-Schneider$^*$},
title = {{H}eterogeneous treatment effects in stratified clinical
trials with time-to-event endpoints.},
journal = {Biometrical journal},
volume = {59},
number = {3},
issn = {0323-3847},
address = {Berlin},
publisher = {Wiley-VCH},
reportid = {DKFZ-2017-01095},
pages = {511 - 530},
year = {2017},
abstract = {When analyzing clinical trials with a stratified
population, homogeneity of treatment effects is a common
assumption in survival analysis. However, in the context of
recent developments in clinical trial design, which aim to
test multiple targeted therapies in corresponding
subpopulations simultaneously, the assumption that there is
no treatment-by-stratum interaction seems inappropriate. It
becomes an issue if the expected sample size of the strata
makes it unfeasible to analyze the trial arms individually.
Alternatively, one might choose as primary aim to prove
efficacy of the overall (targeted) treatment strategy. When
testing for the overall treatment effect, a violation of the
no-interaction assumption renders it necessary to deviate
from standard methods that rely on this assumption. We
investigate the performance of different methods for sample
size calculation and data analysis under heterogeneous
treatment effects. The commonly used sample size formula by
Schoenfeld is compared to another formula by Lachin and
Foulkes, and to an extension of Schoenfeld's formula
allowing for stratification. Beyond the widely used
(stratified) Cox model, we explore the lognormal shared
frailty model, and a two-step analysis approach as potential
alternatives that attempt to adjust for interstrata
heterogeneity. We carry out a simulation study for a trial
with three strata and violations of the no-interaction
assumption. The extension of Schoenfeld's formula to
heterogeneous strata effects provides the most reliable
sample size with respect to desired versus actual power. The
two-step analysis and frailty model prove to be more robust
against loss of power caused by heterogeneous treatment
effects than the stratified Cox model and should be
preferred in such situations.},
cin = {C060},
ddc = {570},
cid = {I:(DE-He78)C060-20160331},
pnm = {313 - Cancer risk factors and prevention (POF3-313)},
pid = {G:(DE-HGF)POF3-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:28263395},
doi = {10.1002/bimj.201600047},
url = {https://inrepo02.dkfz.de/record/120669},
}