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@ARTICLE{Dewi:124343,
author = {D. L. Dewi and S. R. Mohapatra and S. Blanco Cabañes and
I. Adam and L. F. Somarribas Patterson and B. Berdel and M.
Kahloon and L. Thürmann and S. Loth and K. Heilmann$^*$ and
D. Weichenhan$^*$ and O. Mücke$^*$ and I. Heiland and P.
Wimberger$^*$ and J. D. Kuhlmann$^*$ and K.-H. Kellner and
S. Schott and C. Plass$^*$ and M. Platten$^*$ and C.
Gerhäuser$^*$ and S. Trump and C. Opitz$^*$},
title = {{S}uppression of indoleamine-2,3-dioxygenase 1 expression
by promoter hypermethylation in {ER}-positive breast
cancer.},
journal = {OncoImmunology},
volume = {6},
number = {2},
issn = {2162-402X},
address = {Austin, Tex.},
publisher = {Landes Bioscience},
reportid = {DKFZ-2017-01222},
pages = {e1274477 -},
year = {2017},
abstract = {Kynurenine formation by tryptophan-catabolic
indoleamine-2,3-dioxygenase 1 (IDO1) plays a key role in
tumor immune evasion and inhibition of IDO1 is efficacious
in preclinical models of breast cancer. As the response of
breast cancer to immune checkpoint inhibitors may be
limited, a better understanding of the expression of
additional targetable immunomodulatory pathways is of
importance. We therefore investigated the regulation of IDO1
expression in different breast cancer subtypes. We
identified estrogen receptor α (ER) as a negative regulator
of IDO1 expression. Serum kynurenine levels as well as
tumoral IDO1 expression were lower in patients with
ER-positive than ER-negative tumors and an inverse
relationship between IDO1 and estrogen receptor mRNA was
observed across 14 breast cancer data sets. Analysis of
whole genome bisulfite sequencing, 450k, MassARRAY and
pyrosequencing data revealed that the IDO1 promoter is
hypermethylated in ER-positive compared with ER-negative
breast cancer. Reduced induction of IDO1 was also observed
in human ER-positive breast cancer cell lines. IDO1
induction was enhanced upon DNA demethylation in ER-positive
but not in ER-negative cells and methylation of an IDO1
promoter construct reduced IDO1 expression, suggesting that
enhanced methylation of the IDO1 promoter suppresses IDO1 in
ER-positive breast cancer. The association of ER
overexpression with epigenetic downregulation of IDO1
appears to be a particular feature of breast cancer as IDO1
was not suppressed by IDO1 promoter hypermethylation in the
presence of high ER expression in cervical or endometrial
cancer.},
cin = {C010 / G160 / G161 / L301 / L101},
ddc = {610},
cid = {I:(DE-He78)C010-20160331 / I:(DE-He78)G160-20160331 /
I:(DE-He78)G161-20160331 / I:(DE-He78)L301-20160331 /
I:(DE-He78)L101-20160331},
pnm = {317 - Translational cancer research (POF3-317)},
pid = {G:(DE-HGF)POF3-317},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:28344890},
pmc = {pmc:PMC5353999},
doi = {10.1080/2162402X.2016.1274477},
url = {https://inrepo02.dkfz.de/record/124343},
}