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@ARTICLE{Kiesow:126873,
author = {K. Kiesow$^*$ and K. Bennewitz$^*$ and L. G. Miranda$^*$
and S. J. Stoll$^*$ and B. Hartenstein$^*$ and P. Angel$^*$
and J. Kroll$^*$ and M. Schorpp-Kistner$^*$},
title = {{J}unb controls lymphatic vascular development in zebrafish
via mi{R}-182.},
journal = {Scientific reports},
volume = {5},
issn = {2045-2322},
address = {London},
publisher = {Nature Publishing Group},
reportid = {DKFZ-2017-02901},
pages = {15007 -},
year = {2015},
abstract = {JUNB, a subunit of the AP-1 transcription factor complex,
mediates gene regulation in response to a plethora of
extracellular stimuli. Previously, JUNB was shown to act as
a critical positive regulator of blood vessel development
and homeostasis as well as a negative regulator of
proliferation, inflammation and tumour growth. Here, we
demonstrate that the oncogenic miR-182 is a novel JUNB
target. Loss-of-function studies by morpholino-mediated
knockdown and the CRISPR/Cas9 technology identify a novel
function for both JUNB and its target miR-182 in lymphatic
vascular development in zebrafish. Furthermore, we show that
miR-182 attenuates foxo1 expression indicating that strictly
balanced Foxo1 levels are required for proper lymphatic
vascular development in zebrafish. In conclusion, our
findings uncover with the Junb/miR-182/Foxo1 regulatory axis
a novel key player in governing lymphatic vascular
morphogenesis in zebrafish.},
keywords = {Forkhead Box Protein O1 (NLM Chemicals) / Forkhead
Transcription Factors (NLM Chemicals) / FoxO1 protein,
zebrafish (NLM Chemicals) / MicroRNAs (NLM Chemicals) /
Proto-Oncogene Proteins c-jun (NLM Chemicals) / Zebrafish
Proteins (NLM Chemicals)},
cin = {A100 / A190},
ddc = {000},
cid = {I:(DE-He78)A100-20160331 / I:(DE-He78)A190-20160331},
pnm = {321 - Basic Concepts (POF3-321)},
pid = {G:(DE-HGF)POF3-321},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:26458334},
pmc = {pmc:PMC4602192},
doi = {10.1038/srep15007},
url = {https://inrepo02.dkfz.de/record/126873},
}