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@ARTICLE{Zwerger:127890,
author = {M. Zwerger and H. Roschitzki-Voser and R. Zbinden and C.
Denais and H. Herrmann$^*$ and J. Lammerding and M. G.
Grütter and O. Medalia},
title = {{A}ltering lamina assembly reveals lamina-dependent and
-independent functions for {A}-type lamins.},
journal = {Journal of cell science},
volume = {128},
number = {19},
issn = {1477-9137},
address = {Cambridge},
publisher = {Company of Biologists Limited},
reportid = {DKFZ-2017-03912},
pages = {3607 - 3620},
year = {2015},
abstract = {Lamins are intermediate filament proteins that form a
fibrous meshwork, called the nuclear lamina, between the
inner nuclear membrane and peripheral heterochromatin of
metazoan cells. The assembly and incorporation of lamin A/C
into the lamina, as well as their various functions, are
still not well understood. Here, we employed designed
ankyrin repeat proteins (DARPins) as new experimental tools
for lamin research. We screened for DARPins that
specifically bound to lamin A/C, and interfered with lamin
assembly in vitro and with incorporation of lamin A/C into
the native lamina in living cells. The selected DARPins
inhibited lamin assembly and delocalized A-type lamins to
the nucleoplasm without modifying lamin expression levels or
the amino acid sequence. Using these lamin binders, we
demonstrate the importance of proper integration of lamin
A/C into the lamina for nuclear mechanical properties and
nuclear envelope integrity. Finally, our study provides
evidence for cell-type-specific differences in lamin
functions.},
keywords = {Lamin Type A (NLM Chemicals) / Lamin Type B (NLM Chemicals)
/ Lamins (NLM Chemicals)},
cin = {B065},
ddc = {570},
cid = {I:(DE-He78)B065-20160331},
pnm = {312 - Functional and structural genomics (POF3-312)},
pid = {G:(DE-HGF)POF3-312},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:26275827},
pmc = {pmc:PMC4610210},
doi = {10.1242/jcs.171843},
url = {https://inrepo02.dkfz.de/record/127890},
}