% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Zimmer:128704,
author = {P. Zimmer$^*$ and W. Bloch and M. Kieven and L. Lövenich
and J. Lehmann and M. Holthaus and S. Theurich and A.
Schenk},
title = {{S}erotonin {S}hapes the {M}igratory {P}otential of {NK}
{C}ells - {A}n in vitro {A}pproach.},
journal = {International journal of sports medicine},
volume = {38},
number = {11},
issn = {0172-4622},
address = {Stuttgart [u.a.]},
publisher = {Thieme47474},
reportid = {DKFZ-2017-04719},
pages = {857-863},
year = {2017},
abstract = {Increased serotonin (5-HT) levels have been shown to
influence natural killer cell (NK cell) function. Acute
exercise mobilizes and activates NK cells and further
increases serum 5-HT concentrations in a dose-dependent
manner. The aim of this study was to investigate the impact
of different serum 5-HT concentrations on NK cell migratory
potential and cytotoxicity. The human NK cell line KHYG-1
was assigned to 4 conditions, including 3 physiological
concentrations of 5-HT (100, 130 or 170 µg/l 5-HT) and
one control condition. NK cells were analyzed regarding
cytotoxicity, migratory potential and expression of adhesion
molecules. No treatment effect on NK cell cytotoxicity and
expression of integrin subunits was detected. Migratory
potential was increased in a dose dependent manner,
indicating the highest protease activity in cells that were
incubated with 170 µg/l 5-HT (170 µg/l vs. control,
p<0.001, 170 µg/l vs. 100 µg/l, p<0.001; 170 µg/l
vs. 130 µg/l, p=0.003; 130 µg/l vs. control, p<0.001,
130 µg/l vs. 100 µg/l, p<0.001). These results suggest
that elevated 5-HT serum levels play a mediating role in NK
cell function. As exercise has been shown to be involved in
NK cell mobilization and redistribution, the influence of
5-HT should be investigated in ex vivo and in vivo
experiments.},
cin = {G210},
ddc = {610},
cid = {I:(DE-He78)G210-20160331},
pnm = {317 - Translational cancer research (POF3-317)},
pid = {G:(DE-HGF)POF3-317},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:28783845},
doi = {10.1055/s-0043-113042},
url = {https://inrepo02.dkfz.de/record/128704},
}