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@ARTICLE{Larribre:129179,
author = {L. Larribère$^*$ and M. Galach$^*$ and D. Novak$^*$ and K.
Arévalo$^*$ and H. C. Volz$^*$ and H.-J. Stark$^*$ and P.
Boukamp$^*$ and M. Boutros$^*$ and J. Utikal$^*$},
title = {{A}n {RNA}i {S}creen {R}eveals an {E}ssential {R}ole for
{HIPK}4 in {H}uman {S}kin {E}pithelial {D}ifferentiation
from i{PSC}s.},
journal = {Stem cell reports},
volume = {9},
number = {4},
issn = {2213-6711},
address = {Maryland Heights, MO},
publisher = {Cell Press},
reportid = {DKFZ-2017-05184},
pages = {1234 - 1245},
year = {2017},
abstract = {Molecular mechanisms responsible for the development of
human skin epithelial cells are incompletely understood. As
a consequence, the efficiency to establish a pure skin
epithelial cell population from human induced pluripotent
stem cells (hiPSCs) remains poor. Using an approach
including RNAi and high-throughput imaging of early
epithelial cells, we identified candidate kinases involved
in their differentiation from hiPSCs. Among these, we found
HIPK4 to be an important inhibitor of this process. Indeed,
its silencing increased the amount of generated skin
epithelial precursors at an early time point, increased the
amount of generated keratinocytes at a later time point, and
improved growth and differentiation of organotypic cultures,
allowing for the formation of a denser basal layer and
stratification with the expression of several keratins. Our
data bring substantial input regarding regulation of human
skin epithelial differentiation and for improving
differentiation protocols from pluripotent stem cells.},
cin = {G300 / B110 / A110},
ddc = {610},
cid = {I:(DE-He78)G300-20160331 / I:(DE-He78)B110-20160331 /
I:(DE-He78)A110-20160331},
pnm = {317 - Translational cancer research (POF3-317)},
pid = {G:(DE-HGF)POF3-317},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:28966120},
pmc = {pmc:PMC5639458},
doi = {10.1016/j.stemcr.2017.08.023},
url = {https://inrepo02.dkfz.de/record/129179},
}