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@ARTICLE{Pajtler:130295,
author = {K. Pajtler$^*$ and S. Tippelt and N. Siegler and S.
Reichling and M. Zimmermann and R. Mikasch and U. Bode and
A. Gnekow and T. Pietsch and M. Benesch and S. Rutkowski and
G. Fleischhack},
title = {{I}ntraventricular etoposide safety and toxicity profile in
children and young adults with refractory or recurrent
malignant brain tumors.},
journal = {Journal of neuro-oncology},
volume = {128},
number = {3},
issn = {1573-7373},
address = {Dordrecht [u.a.]},
publisher = {Springer Science + Business Media B.V},
reportid = {DKFZ-2017-05374},
pages = {463 - 471},
year = {2016},
abstract = {Systemic administration of etoposide is effective in
treating metastatic, recurrent or refractory brain tumors,
but penetration into the cerebrospinal fluid is extremely
poor. This study was designed to determine the safety and
toxicity profile of intraventricular etoposide
administration and was affiliated with the prospective,
multicenter, nonblinded, nonrandomized, multi-armed
HIT-REZ-97 trial. The study enrolled 68 patients, aged
1.1-34.6 (median age 11 years). Adverse events that could
possibly be related to intraventricular etoposide therapy
were documented and analyzed. Intraventricular etoposide was
simultaneously administered with either oral or intravenous
chemotherapy in 426 courses according to three major
schedules varying in dosing (0.25-1 mg), frequency of
administration (bolus injection, every 12 or 24 h), course
duration (5-10 days) and length of interval between courses
(2-5 weeks). Potential treatment-related adverse effects
included transient headache, seizures, infection of the
reservoir, nausea and neuropsychological symptoms.
Hematological side effects were not observed. One patient,
with history of multiple prior therapies, who received
long-term intraventricular and oral etoposide treatment
developed acute myeloid leukemia as a secondary malignancy.
Overall intraventricular etoposide is well tolerated. The
results of this study have warranted a phase II trial to
determine the effectiveness of this regimen in disease
stages with very limited therapeutic options.},
cin = {B062},
ddc = {610},
cid = {I:(DE-He78)B062-20160331},
pnm = {312 - Functional and structural genomics (POF3-312)},
pid = {G:(DE-HGF)POF3-312},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:27147083},
doi = {10.1007/s11060-016-2133-x},
url = {https://inrepo02.dkfz.de/record/130295},
}