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@ARTICLE{Bchele:132695,
author = {G. Büchele and K. P. Günther and H. Brenner$^*$ and W.
Puhl and T. Stürmer and D. Rothenbacher and R. E. Brenner},
title = {{O}steoarthritis-patterns, cardio-metabolic risk factors
and risk of all-cause mortality: 20 years follow-up in
patients after hip or knee replacement.},
journal = {Scientific reports},
volume = {8},
number = {1},
issn = {2045-2322},
address = {London},
publisher = {Nature Publishing Group},
reportid = {DKFZ-2018-00352},
pages = {5253},
year = {2018},
abstract = {Osteoarthritis (OA) is a common musculoskeletal disorder
and occur in different patterns. However, its impact on
long-term all-cause-mortality is inconclusive.Investigate
20-year all-cause-mortality in patients with hip/knee
arthroplasty (recruited 1995/1996, N = 809) from the Ulm
Osteoarthritis Study-cohort, in comparison to general
population. Furthermore, to enlighten the triangle between
baseline life-style and cardio-metabolic risk factors,
phenotypic OA-patterns (laterality, generalization, cause)
and all-cause-mortality. Mortality was assessed during 20
years follow-up. Standardized mortality ratios (SMR),
adjusted odds ratios and hazard ratios (aHR) were
calculated. After five years cohort-mortality was reduced
compared to the general population, however 20 years later
assimilated (SMR = 1.11; $95\%-CI$ 0.73-1.49).
OA-patterns were associated with age, cholesterol, and
overweight/obesity. In comparison to primary OA decreased
mortality was observed for patients with secondary OA
(aHR = 0.76; $95\%-CI$ 0.61-0.95) adjusted for age,
smoking, overweight/obesity, diabetes, hypertension, cardiac
insufficiency, uric acid, and lower cholesterol. There was
no increased mortality in patients after 20 years follow-up
compared to general population. Significantly decreased
mortality in secondary compared to primary OA suggests a
subtype-specific involvement of systemic co-factors in
determination of all-cause-mortality. Because
cardio-metabolic risk factors were associated with increased
risk of bilateral OA and lower long-term survival, those
risk factors should be consequently targeted in
OA-patients.},
cin = {C070 / G110},
ddc = {000},
cid = {I:(DE-He78)C070-20160331 / I:(DE-He78)G110-20160331},
pnm = {323 - Metabolic Dysfunction as Risk Factor (POF3-323)},
pid = {G:(DE-HGF)POF3-323},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:29588472},
pmc = {pmc:PMC5869736},
doi = {10.1038/s41598-018-23573-2},
url = {https://inrepo02.dkfz.de/record/132695},
}