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@ARTICLE{Grbner:132715,
author = {S. Gröbner$^*$ and B. Worst$^*$ and J. Weischenfeldt and
I. Buchhalter$^*$ and K. Kleinheinz$^*$ and V. A. Rudneva
and P. Johann$^*$ and G. P. Balasubramanian and M.
Segura-Wang and S. Brabetz$^*$ and S. Bender and B.
Hutter$^*$ and D. Sturm$^*$ and E. Pfaff$^*$ and D.
Hübschmann$^*$ and G. Zipprich$^*$ and M. Heinold$^*$ and
J. Eils$^*$ and C. Lawerenz$^*$ and S. Erkek$^*$ and S.
Lambo$^*$ and S. Waszak and C. Blattmann and A. Borkhardt
and M. Kuhlen and A. Eggert and S. Fulda and M. Gessler and
J. Wegert and R. Kappler and D. Baumhoer and S. Burdach and
R. Kirschner-Schwabe and U. Kontny and A. E. Kulozik and D.
Lohmann and S. Hettmer and C. Eckert and S. Bielack and M.
Nathrath and C. Niemeyer and G. H. Richter and J.
Schulte$^*$ and R. Siebert and F. Westermann$^*$ and J. J.
Molenaar and G. Vassal and H. Witt$^*$ and B. Burkhardt and
C. P. Kratz and O. Witt$^*$ and C. M. van Tilburg and C. M.
Kramm and G. Fleischhack and U. Dirksen and S. Rutkowski and
M. Frühwald and K. von Hoff and S. Wolf$^*$ and T.
Klingebiel and E. Koscielniak and P. Landgraf and J. Koster
and A. C. Resnick and J. Zhang and Y. Liu and X. Zhou and A.
J. Waanders and D. A. Zwijnenburg and P. Raman and B.
Brors$^*$ and U. Weber$^*$ and P. A. Northcott and K.
Pajtler$^*$ and M. Kool$^*$ and R. M. Piro and J. O. Korbel
and M. Schlesner$^*$ and R. Eils$^*$ and D. Jones$^*$ and P.
Lichter$^*$ and L. Chavez$^*$ and M. Zapatka$^*$ and S.
Pfister$^*$},
collaboration = {I. P. Project and I. M. Project},
title = {{T}he landscape of genomic alterations across childhood
cancers.},
journal = {Nature},
volume = {555},
number = {7696},
issn = {1476-4687},
address = {London [u.a.]},
publisher = {Nature Publ. Group},
reportid = {DKFZ-2018-00369},
pages = {321 - 327},
year = {2018},
abstract = {Pan-cancer analyses that examine commonalities and
differences among various cancer types have emerged as a
powerful way to obtain novel insights into cancer biology.
Here we present a comprehensive analysis of genetic
alterations in a pan-cancer cohort including 961 tumours
from children, adolescents, and young adults, comprising 24
distinct molecular types of cancer. Using a standardized
workflow, we identified marked differences in terms of
mutation frequency and significantly mutated genes in
comparison to previously analysed adult cancers. Genetic
alterations in 149 putative cancer driver genes separate the
tumours into two classes: small mutation and
structural/copy-number variant (correlating with germline
variants). Structural variants, hyperdiploidy, and
chromothripsis are linked to TP53 mutation status and
mutational signatures. Our data suggest that $7-8\%$ of the
children in this cohort carry an unambiguous predisposing
germline variant and that nearly $50\%$ of paediatric
neoplasms harbour a potentially druggable event, which is
highly relevant for the design of future clinical trials.},
cin = {B062 / L101 / B080 / G200 / B087 / G340 / W190 / B060 /
B240},
ddc = {070},
cid = {I:(DE-He78)B062-20160331 / I:(DE-He78)L101-20160331 /
I:(DE-He78)B080-20160331 / I:(DE-He78)G200-20160331 /
I:(DE-He78)B087-20160331 / I:(DE-He78)G340-20160331 /
I:(DE-He78)W190-20160331 / I:(DE-He78)B060-20160331 /
I:(DE-He78)B240-20160331},
pnm = {312 - Functional and structural genomics (POF3-312)},
pid = {G:(DE-HGF)POF3-312},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:29489754},
doi = {10.1038/nature25480},
url = {https://inrepo02.dkfz.de/record/132715},
}