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@ARTICLE{Meidtner:132893,
author = {K. Meidtner and C. Podmore and J. Kröger and Y. T. van der
Schouw and B. Bendinelli and C. Agnoli and L. Arriola and A.
Barricarte and H. Boeing and A. J. Cross and C. Dow and K.
Ekblom and G. Fagherazzi and P. W. Franks and M. J. Gunter
and J. M. Huerta and P. Jakszyn and M. Jenab and V.
Katzke$^*$ and T. J. Key and K. T. Khaw and T. Kühn$^*$ and
C. Kyrø and F. R. Mancini and O. Melander and P. M. Nilsson
and K. Overvad and D. Palli and S. Panico and J. R. Quirós
and M. Rodríguez-Barranco and C. Sacerdote and I. Sluijs
and M. Stepien and A. Tjonneland and R. Tumino and N. G.
Forouhi and S. J. Sharp and C. Langenberg and M. B. Schulze
and E. Riboli and N. J. Wareham},
title = {{I}nteraction of {D}ietary and {G}enetic {F}actors
{I}nfluencing {B}ody {I}ron {S}tatus and {R}isk of {T}ype 2
{D}iabetes {W}ithin the {EPIC}-{I}nter{A}ct {S}tudy.},
journal = {Diabetes care},
volume = {41},
number = {2},
issn = {1935-5548},
address = {Alexandria, Va.},
publisher = {Assoc.},
reportid = {DKFZ-2018-00535},
pages = {277 - 285},
year = {2018},
abstract = {Meat intake has been consistently shown to be positively
associated with incident type 2 diabetes. Part of that
association may be mediated by body iron status, which is
influenced by genetic factors. We aimed to test for
interactions of genetic and dietary factors influencing body
iron status in relation to the risk of incident type 2
diabetes.The case-cohort comprised 9,347 case subjects and
12,301 subcohort participants from eight European countries.
Single nucleotide polymorphisms (SNPs) were selected from
genome-wide association studies on iron status biomarkers
and candidate gene studies. A ferritin-related gene score
was constructed. Multiplicative and additive interactions of
heme iron and SNPs as well as the gene score were evaluated
using Cox proportional hazards regression.Higher heme iron
intake (per 1 SD) was associated with higher ferritin levels
(β = 0.113 $[95\%$ CI 0.082; 0.144]), but not with
transferrin (-0.019 [-0.043; 0.006]) or transferrin
saturation (0.016 [-0.006; 0.037]). Five SNPs located in
four genes (rs1799945 [HFE H63D], rs1800562 [HFE C282Y],
rs236918 [PCK7], rs744653 [SLC40A1], and rs855791 [TMPRSS6
V736A]) were associated with ferritin. We did not detect an
interaction of heme iron and the gene score on the risk of
diabetes in the overall study population (Padd = 0.16, Pmult
= 0.21) but did detect a trend toward a negative interaction
in men (Padd = 0.04, Pmult = 0.03).We found no convincing
evidence that the interplay of dietary and genetic factors
related to body iron status associates with type 2 diabetes
risk above the level expected from the sum or product of the
two individual exposures.},
cin = {C020},
ddc = {610},
cid = {I:(DE-He78)C020-20160331},
pnm = {313 - Cancer risk factors and prevention (POF3-313)},
pid = {G:(DE-HGF)POF3-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:29167213},
doi = {10.2337/dc17-1080},
url = {https://inrepo02.dkfz.de/record/132893},
}