TY - JOUR
AU - Mackay, Alan
AU - Burford, Anna
AU - Molinari, Valeria
AU - Jones, David
AU - Izquierdo, Elisa
AU - Brouwer-Visser, Jurriaan
AU - Giangaspero, Felice
AU - Haberler, Christine
AU - Pietsch, Torsten
AU - Jacques, Thomas S
AU - Figarella-Branger, Dominique
AU - Rodriguez, Daniel
AU - Morgan, Paul S
AU - Raman, Pichai
AU - Waanders, Angela J
AU - Resnick, Adam C
AU - Massimino, Maura
AU - Garrè, Maria Luisa
AU - Smith, Helen
AU - Capper, David
AU - Pfister, Stefan
AU - Würdinger, Thomas
AU - Tam, Rachel
AU - Garcia, Josep
AU - Thakur, Meghna Das
AU - Vassal, Gilles
AU - Grill, Jacques
AU - Jaspan, Tim
AU - Varlet, Pascale
AU - Jones, Chris
TI - Molecular, Pathological, Radiological, and Immune Profiling of Non-brainstem Pediatric High-Grade Glioma from the HERBY Phase II Randomized Trial.
JO - Cancer cell
VL - 33
IS - 5
SN - 1535-6108
CY - Cambridge, Mass.
PB - Cell Press
M1 - DKFZ-2018-00623
SP - 829 - 842.e5
PY - 2018
AB - The HERBY trial was a phase II open-label, randomized, multicenter trial evaluating bevacizumab (BEV) in addition to temozolomide/radiotherapy in patients with newly diagnosed non-brainstem high-grade glioma (HGG) between the ages of 3 and 18 years. We carried out comprehensive molecular analysis integrated with pathology, radiology, and immune profiling. In post-hoc subgroup analysis, hypermutator tumors (mismatch repair deficiency and somatic POLE/POLD1 mutations) and those biologically resembling pleomorphic xanthoastrocytoma ([PXA]-like, driven by BRAF_V600E or NF1 mutation) had significantly more CD8+ tumor-infiltrating lymphocytes, and longer survival with the addition of BEV. Histone H3 subgroups (hemispheric G34R/V and midline K27M) had a worse outcome and were immune cold. Future clinical trials will need to take into account the diversity represented by the term 'HGG' in the pediatric population.
LB - PUB:(DE-HGF)16
C6 - pmid:29763623
C2 - pmc:PMC5956280
DO - DOI:10.1016/j.ccell.2018.04.004
UR - https://inrepo02.dkfz.de/record/134833
ER -