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@ARTICLE{Kickingereder:134853,
author = {P. Kickingereder and U. Neuberger and D. Bonekamp$^*$ and
P. L. Piechotta and M. Götz$^*$ and A. Wick and M. Sill$^*$
and A. Kratz$^*$ and R. T. Shinohara and D. Jones$^*$ and A.
Radbruch$^*$ and J. Muschelli and A. Unterberg and J.
Debus$^*$ and H.-P. Schlemmer$^*$ and C. Herold-Mende$^*$
and S. Pfister$^*$ and A. von Deimling$^*$ and W. Wick$^*$
and D. Capper$^*$ and K. Maier-Hein$^*$ and M. Bendszus},
title = {{R}adiomic subtyping improves disease stratification beyond
key molecular, clinical, and standard imaging
characteristics in patients with glioblastoma.},
journal = {Neuro-Oncology},
volume = {20},
number = {6},
issn = {1523-5866},
address = {Oxford},
publisher = {Oxford Univ. Press},
reportid = {DKFZ-2018-00643},
pages = {848 - 857},
year = {2018},
abstract = {The purpose of this study was to analyze the potential of
radiomics for disease stratification beyond key molecular,
clinical, and standard imaging features in patients with
glioblastoma.Quantitative imaging features (n = 1043) were
extracted from the multiparametric MRI of 181 patients with
newly diagnosed glioblastoma prior to standard-of-care
treatment (allocated to a discovery and a validation set,
2:1 ratio). A subset of 386/1043 features were identified as
reproducible (in an independent MRI test-retest cohort) and
selected for analysis. A penalized Cox model with 10-fold
cross-validation (Coxnet) was fitted on the discovery set to
construct a radiomic signature for predicting
progression-free and overall survival (PFS and OS). The
incremental value of a radiomic signature beyond molecular
(O6-methylguanine-DNA methyltransferase [MGMT] promoter
methylation, DNA methylation subgroups), clinical (patient's
age, KPS, extent of resection, adjuvant treatment), and
standard imaging parameters (tumor volumes) for stratifying
PFS and OS was assessed with multivariate Cox models
(performance quantified with prediction error curves).The
radiomic signature (constructed from 8/386 features
identified through Coxnet) increased the prediction accuracy
for PFS and OS (in both discovery and validation sets)
beyond the assessed molecular, clinical, and standard
imaging parameters (P ≤ 0.01). Prediction errors decreased
by $36\%$ for PFS and $37\%$ for OS when adding the radiomic
signature (compared with $29\%$ and $27\%,$ respectively,
with molecular + clinical features alone). The radiomic
signature was-along with MGMT status-the only parameter with
independent significance on multivariate analysis (P ≤
0.01).Our study stresses the role of integrating radiomics
into a multilayer decision framework with key molecular and
clinical features to improve disease stratification and to
potentially advance personalized treatment of patients with
glioblastoma.},
cin = {E010 / E230 / E132 / C060 / G380 / B062 / L101 / G370 /
E050},
ddc = {610},
cid = {I:(DE-He78)E010-20160331 / I:(DE-He78)E230-20160331 /
I:(DE-He78)E132-20160331 / I:(DE-He78)C060-20160331 /
I:(DE-He78)G380-20160331 / I:(DE-He78)B062-20160331 /
I:(DE-He78)L101-20160331 / I:(DE-He78)G370-20160331 /
I:(DE-He78)E050-20160331},
pnm = {315 - Imaging and radiooncology (POF3-315)},
pid = {G:(DE-HGF)POF3-315},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:29036412},
pmc = {pmc:PMC5961168},
doi = {10.1093/neuonc/nox188},
url = {https://inrepo02.dkfz.de/record/134853},
}