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@ARTICLE{Koelsche:136745,
author = {C. Koelsche$^*$ and M. Mynarek and D. Schrimpf$^*$ and L.
Bertero and J. Serrano and F. Sahm$^*$ and D. E. Reuss$^*$
and Y. Hou and D. Baumhoer and C. Vokuhl and U. Flucke and
I. Petersen and W. Brück and S. Rutkowski and S. C.
Zambrano and J. L. Garcia Leon and R. Y. Diaz Coronado and
M. Gessler and O. M. Tirado and J. Mora and J. Alonso and X.
Garcia Del Muro and M. Esteller and D. Sturm$^*$ and J.
Ecker$^*$ and T. Milde$^*$ and S. Pfister$^*$ and A.
Korshunov$^*$ and M. Snuderl and G. Mechtersheimer and U.
Schüller and D. Jones$^*$ and A. von Deimling$^*$},
title = {{P}rimary intracranial spindle cell sarcoma with
rhabdomyosarcoma-like features share a highly distinct
methylation profile and {DICER}1 mutations.},
journal = {Acta neuropathologica},
volume = {136},
number = {2},
issn = {1432-0533},
address = {Berlin},
publisher = {Springer},
reportid = {DKFZ-2018-01183},
pages = {327 - 337},
year = {2018},
abstract = {Patients with DICER1 predisposition syndrome have an
increased risk to develop pleuropulmonary blastoma, cystic
nephroma, embryonal rhabdomyosarcoma, and several other rare
tumor entities. In this study, we identified 22 primary
intracranial sarcomas, including 18 in pediatric patients,
with a distinct methylation signature detected by
array-based DNA-methylation profiling. In addition, two
uterine rhabdomyosarcomas sharing identical features were
identified. Gene panel sequencing of the 22 intracranial
sarcomas revealed the almost unifying feature of DICER1
hotspot mutations (21/22; $95\%)$ and a high frequency of
co-occurring TP53 mutations (12/22; $55\%).$ In addition,
17/22 $(77\%)$ sarcomas exhibited alterations in the
mitogen-activated protein kinase pathway, most frequently
affecting the mutational hotspots of KRAS (8/22; $36\%)$ and
mutations or deletions of NF1 (7/22; $32\%),$ followed by
mutations of FGFR4 (2/22; $9\%),$ NRAS (2/22; $9\%),$ and
amplification of EGFR (1/22; $5\%).$ A germline DICER1
mutation was detected in two of five cases with
constitutional DNA available. Notably, none of the patients
showed evidence of a cancer-related syndrome at the time of
diagnosis. In contrast to the genetic findings, the
morphological features of these tumors were less
distinctive, although rhabdomyoblasts or rhabdomyoblast-like
cells could retrospectively be detected in all cases. The
identified combination of genetic events indicates a
relationship between the intracranial tumors analyzed and
DICER1 predisposition syndrome-associated sarcomas such as
embryonal rhabdomyosarcoma or the recently described group
of anaplastic sarcomas of the kidney. However, the
intracranial tumors in our series were initially interpreted
to represent various tumor types, but rhabdomyosarcoma was
not among the typical differential diagnoses considered.
Given the rarity of intracranial sarcomas, this molecularly
clearly defined group comprises a considerable fraction
thereof. We therefore propose the designation 'spindle cell
sarcoma with rhabdomyosarcoma-like features, DICER1 mutant'
for this intriguing group.},
cin = {G380 / L101 / B062 / G340 / G702},
ddc = {610},
cid = {I:(DE-He78)G380-20160331 / I:(DE-He78)L101-20160331 /
I:(DE-He78)B062-20160331 / I:(DE-He78)G340-20160331 /
I:(DE-He78)G702-20160331},
pnm = {317 - Translational cancer research (POF3-317)},
pid = {G:(DE-HGF)POF3-317},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:29881993},
doi = {10.1007/s00401-018-1871-6},
url = {https://inrepo02.dkfz.de/record/136745},
}