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@ARTICLE{Murphy:137000,
author = {N. Murphy and D. Achaintre and R. Zamora-Ros and M. Jenab
and M.-C. Boutron-Ruault and F. Carbonnel and I. Savoye and
R. Kaaks$^*$ and T. Kühn$^*$ and H. Boeing and K.
Aleksandrova and A. Tjønneland and C. Kyrø and K. Overvad
and J. R. Quirós and M.-J. Sánchez and J. M. Altzibar and
J. María Huerta and A. Barricarte and K.-T. Khaw and K. E.
Bradbury and A. Perez-Cornago and A. Trichopoulou and A.
Karakatsani and E. Peppa and D. Palli and S. Grioni and R.
Tumino and C. Sacerdote and S. Panico and H. B. A.
Bueno-de-Mesquita and P. H. Peeters and M. Rutegård and I.
Johansson and H. Freisling and H. Noh and A. J. Cross and P.
Vineis and K. Tsilidis and M. J. Gunter and A. Scalbert},
title = {{A} prospective evaluation of plasma polyphenol levels and
colon cancer risk.},
journal = {International journal of cancer},
volume = {143},
number = {7},
issn = {0020-7136},
address = {Bognor Regis},
publisher = {Wiley-Liss},
reportid = {DKFZ-2018-01428},
pages = {1620 - 1631},
year = {2018},
abstract = {Polyphenols have been shown to exert biological activity in
experimental models of colon cancer; however, human data
linking specific polyphenols to colon cancer is limited. We
assessed the relationship between pre-diagnostic plasma
polyphenols and colon cancer risk in a case-control study
nested within the European Prospective Investigation into
Cancer and Nutrition study. Using high pressure liquid
chromatography coupled to tandem mass spectrometry, we
measured concentrations of 35 polyphenols in plasma from 809
incident colon cancer cases and 809 matched controls. We
used multivariable adjusted conditional logistic regression
models that included established colon cancer risk factors.
The false discovery rate (qvalues ) was computed to control
for multiple comparisons. All statistical tests were
two-sided. After false discovery rate correction and in
continuous log2 -transformed multivariable models, equol
(odds ratio [OR] per log2 -value, 0.86, $95\%$ confidence
interval $[95\%$ CI] = 0.79-0.93; qvalue = 0.01)
and homovanillic acid (OR per log2 -value, 1.46, $95\%$
CI = 1.16-1.84; qvalue = 0.02) were associated with
colon cancer risk. Comparing extreme fifths, equol
concentrations were inversely associated with colon cancer
risk (OR = 0.61, $95\%$ CI = 0.41-0.91, ptrend
= 0.003), while homovanillic acid concentrations were
positively associated with colon cancer development
(OR = 1.72, $95\%$ CI = 1.17-2.53, ptrend
< 0.0001). No heterogeneity for these associations was
observed by sex and across other colon cancer risk factors.
The remaining polyphenols were not associated with colon
cancer risk. Higher equol concentrations were associated
with lower risk, and higher homovanillic acid concentrations
were associated with greater risk of colon cancer. These
findings support a potential role for specific polyphenols
in colon tumorigenesis.},
cin = {C020},
ddc = {610},
cid = {I:(DE-He78)C020-20160331},
pnm = {313 - Cancer risk factors and prevention (POF3-313)},
pid = {G:(DE-HGF)POF3-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:29696648},
doi = {10.1002/ijc.31563},
url = {https://inrepo02.dkfz.de/record/137000},
}