TY - JOUR
AU - Gonzalez-Duque, Sergio
AU - Azoury, Marie Eliane
AU - Colli, Maikel L
AU - Afonso, Georgia
AU - Turatsinze, Jean-Valery
AU - Nigi, Laura
AU - Lalanne, Ana Ines
AU - Sebastiani, Guido
AU - Carré, Alexia
AU - Pinto, Sheena
AU - Culina, Slobodan
AU - Corcos, Noémie
AU - Bugliani, Marco
AU - Marchetti, Piero
AU - Armanet, Mathieu
AU - Diedisheim, Marc
AU - Kyewski, Bruno
AU - Steinmetz, Lars M
AU - Buus, Søren
AU - You, Sylvaine
AU - Dubois-Laforgue, Daniele
AU - Larger, Etienne
AU - Beressi, Jean-Paul
AU - Bruno, Graziella
AU - Dotta, Francesco
AU - Scharfmann, Raphael
AU - Eizirik, Decio L
AU - Verdier, Yann
AU - Vinh, Joelle
AU - Mallone, Roberto
TI - Conventional and Neo-antigenic Peptides Presented by β Cells Are Targeted by Circulating Naïve CD8+ T Cells in Type 1 Diabetic and Healthy Donors.
JO - Cell metabolism
VL - 28
IS - 6
SN - 1550-4131
CY - Cambridge, Mass.
PB - Cell Press
M1 - DKFZ-2018-02268
SP - 946 - 960.e6
PY - 2018
AB - Although CD8+ T-cell-mediated autoimmune β cell destruction occurs in type 1 diabetes (T1D), the target epitopes processed and presented by β cells are unknown. To identify them, we combined peptidomics and transcriptomics strategies. Inflammatory cytokines increased peptide presentation in vitro, paralleling upregulation of human leukocyte antigen (HLA) class I expression. Peptide sources featured several insulin granule proteins and all known β cell antigens, barring islet-specific glucose-6-phosphatase catalytic subunit-related protein. Preproinsulin yielded HLA-A2-restricted epitopes previously described. Secretogranin V and its mRNA splice isoform SCG5-009, proconvertase-2, urocortin-3, the insulin gene enhancer protein ISL-1, and an islet amyloid polypeptide transpeptidation product emerged as antigens processed into HLA-A2-restricted epitopes, which, as those already described, were recognized by circulating naive CD8+ T cells in T1D and healthy donors and by pancreas-infiltrating cells in T1D donors. This peptidome opens new avenues to understand antigen processing by β cells and for the development of T cell biomarkers and tolerogenic vaccination strategies.
LB - PUB:(DE-HGF)16
C6 - pmid:30078552
DO - DOI:10.1016/j.cmet.2018.07.007
UR - https://inrepo02.dkfz.de/record/142038
ER -