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@ARTICLE{Katsanovskaja:143329,
author = {K. Katsanovskaja and T. Driver and R. Pipkorn$^*$ and M.
Edelson-Averbukh},
title = {{N}egative {I}on {M}ode {C}ollision-{I}nduced
{D}issociation for {A}nalysis of {P}rotein {A}rginine
{M}ethylation.},
journal = {Journal of the American Society for Mass Spectrometry},
volume = {30},
number = {7},
issn = {1879-1123},
address = {New York [u.a.]},
publisher = {Springer},
reportid = {DKFZ-2019-00919},
pages = {1229-1241},
year = {2019},
abstract = {Arginine methylation is a common protein post-translational
modification (PTM) that plays a key role in eukaryotic
cells. Three distinct types of this modification are found
in mammals: asymmetric Nη1Nη1-dimethylarginine (aDMA),
symmetric Nη1Nη2-dimethylarginine (sDMA), and an
intermediate Nη1-monomethylarginine (MMA). Elucidation of
regulatory mechanisms of arginine methylation in living
organisms requires precise information on both the type of
the modified residues and their location inside the protein
amino acid sequences. Despite mass spectrometry (MS) being
the method of choice for analysis of multiple protein PTMs,
unambiguous characterization of protein arginine methylation
may not be always straightforward. Indeed, frequent internal
basic residues of Arg methylated tryptic peptides hamper
their sequencing under positive ion mode collision-induced
dissociation (CID), the standardly used tandem mass
spectrometry method, while the relative stability of the
aDMA and sDMA side chains under alternative non-ergodic
electron-based fragmentation techniques, electron-capture
and electron transfer dissociations (ECD and ETD), may
impede differentiation between the isobaric residues. Here,
for the first time, we demonstrate the potential of the
negative ion mode collision-induced dissociation MS for
analysis of protein arginine methylation and present data
revealing that the negative polarity approach can deliver
both an unambiguous identification of the arginine
methylation type and extensive information on the modified
peptide sequences.},
cin = {D015},
ddc = {530},
cid = {I:(DE-He78)D015-20160331},
pnm = {314 - Tumor immunology (POF3-314)},
pid = {G:(DE-HGF)POF3-314},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:30915654},
doi = {10.1007/s13361-019-02176-9},
url = {https://inrepo02.dkfz.de/record/143329},
}