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@ARTICLE{Assi:143787,
author = {N. Assi and S. Rinaldi and V. Viallon and S. G. Dashti and
L. Dossus and A. Fournier and I. Cervenka and M. Kvaskoff
and R. Turzanski-Fortner$^*$ and M. Bergmann and H. Boeing
and S. Panico and F. Ricceri and D. Palli and R. Tumino and
S. Grioni and M. J. Sánchez Pérez and M.-D. Chirlaque and
C. Bonet and A. B. Gurrea and P. Amiano Etxezarreta and S.
Merino and H. B. Bueno de Mesquita and C. H. van Gils and C.
Onland-Moret and A. Tjønneland and K. Overvad and A.
Trichopoulou and G. Martimianaki and A. Karakatsani and T.
Key and S. Christakoudi and M. Ellingjord-Dale and K.
Tsilidis and E. Riboli and R. Kaaks$^*$ and M. J. Gunter and
P. Ferrari},
title = {{M}ediation analysis of the alcohol-postmenopausal breast
cancer relationship by sex hormones in the {EPIC} cohort.},
journal = {International journal of cancer},
volume = {146},
number = {3},
issn = {1097-0215},
address = {Bognor Regis},
publisher = {Wiley-Liss},
reportid = {DKFZ-2019-01356},
pages = {759-768},
year = {2020},
note = {2020 Feb 1;146(3):759-768},
abstract = {Alcohol consumption is associated with higher risk of
breast cancer (BC); however, the biological mechanisms
underlying this association are not fully elucidated,
particularly the extent to which this relationship is
mediated by sex hormone levels. Circulating concentrations
of estradiol, testosterone, their free fractions and
sex-hormone binding globulin (SHBG), were examined in 430
incident BC cases and 645 matched controls among
alcohol-consuming postmenopausal women nested within the
European Prospective Investigation into Cancer and
Nutrition. Mediation analysis was applied to assess whether
individual hormone levels mediated the relationship between
alcohol intake and BC risk. An alcohol-related hormonal
signature, obtained by partial least square (PLS)
regression, was evaluated as a potential mediator. Total
(TE), natural direct and natural indirect effects (NIE) were
estimated. Alcohol intake was positively associated with
overall BC risk and specifically with estrogen
receptor-positive tumors with respectively
$TE = 1.17(95\%CI:$ 1.01,1.35) and 1.36(1.08,1.70) for a
1-standard deviation (1-SD) increase of intake. There was no
evidence of mediation by sex steroids or SHBG separately
except for a weak indirect effect through free estradiol
where NIE = 1.03(1.00,1.06). However, an alcohol-related
hormonal signature negatively associated with SHBG and
positively with estradiol and testosterone was associated
with BC risk (odds ratio [OR] = 1.25 [1.07,1.47]) for a
1-SD higher PLS score, and had a statistically significant
NIE accounting for a mediated proportion of $24\%.$ There
was limited evidence of mediation of the alcohol-BC
association by individual sex hormones. However, a hormonal
signature, reflecting lower levels of SHBG and higher levels
of sex steroids, mediated a substantial proportion of the
association.},
cin = {C020},
ddc = {610},
cid = {I:(DE-He78)C020-20160331},
pnm = {313 - Cancer risk factors and prevention (POF3-313)},
pid = {G:(DE-HGF)POF3-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:30968961},
doi = {10.1002/ijc.32324},
url = {https://inrepo02.dkfz.de/record/143787},
}