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@ARTICLE{Geijsen:144343,
author = {A. J. M. R. Geijsen and S. Brezina and P. Keski-Rahkonen
and A. Baierl and T. Bachleitner-Hofmann and M. M. Bergmann
and J. Boehm and H. Brenner$^*$ and J. Chang-Claude$^*$ and
F. J. B. van Duijnhoven and B. Gigic and T. Gumpenberger and
P. Hofer and M. Hoffmeister$^*$ and A. N. Holowatyj and J.
Karner-Hanusch and D. E. Kok and G. Leeb and A. Ulvik and N.
Robinot and J. Ose and A. Stift and P. Schrotz-King$^*$ and
A. B. Ulrich and P. M. Ueland and E. Kampman and A. Scalbert
and N. Habermann$^*$ and A. Gsur and C. M. Ulrich},
title = {{P}lasma metabolites associated with colorectal cancer: {A}
discovery-replication strategy.},
journal = {International journal of cancer},
volume = {145},
number = {5},
issn = {0020-7136},
address = {Bognor Regis},
publisher = {Wiley-Liss},
reportid = {DKFZ-2019-01796},
pages = {1221 - 1231},
year = {2019},
abstract = {Colorectal cancer is known to arise from multiple
tumorigenic pathways; however, the underlying mechanisms
remain not completely understood. Metabolomics is becoming
an increasingly popular tool in assessing biological
processes. Previous metabolomics research focusing on
colorectal cancer is limited by sample size and did not
replicate findings in independent study populations to
verify robustness of reported findings. Here, we performed a
ultrahigh performance liquid chromatography-quadrupole
time-of-flight mass spectrometry (UHPLC-QTOF-MS) screening
on EDTA plasma from 268 colorectal cancer patients and 353
controls using independent discovery and replication sets
from two European cohorts (ColoCare Study: n = 180
patients/n = 153 controls; the Colorectal Cancer Study
of Austria (CORSA) n = 88 patients/n = 200
controls), aiming to identify circulating plasma metabolites
associated with colorectal cancer and to improve knowledge
regarding colorectal cancer etiology. Multiple logistic
regression models were used to test the association between
disease state and metabolic features. Statistically
significant associated features in the discovery set were
taken forward and tested in the replication set to assure
robustness of our findings. All models were adjusted for
sex, age, BMI and smoking status and corrected for multiple
testing using False Discovery Rate. Demographic and clinical
data were abstracted from questionnaires and medical
records.},
cin = {C120 / C070 / L101 / C020},
ddc = {610},
cid = {I:(DE-He78)C120-20160331 / I:(DE-He78)C070-20160331 /
I:(DE-He78)L101-20160331 / I:(DE-He78)C020-20160331},
pnm = {313 - Cancer risk factors and prevention (POF3-313)},
pid = {G:(DE-HGF)POF3-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:30665271},
pmc = {pmc:PMC6614008},
doi = {10.1002/ijc.32146},
url = {https://inrepo02.dkfz.de/record/144343},
}