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@ARTICLE{Ehrlich:144478,
author = {F. Ehrlich and M. Laggner and L. Langbein$^*$ and P. Burger
and A. Pollreisz and E. Tschachler and L. Eckhart},
title = {{C}omparative genomics suggests loss of keratin {K}24 in
three evolutionary lineages of mammals.},
journal = {Scientific reports},
volume = {9},
number = {1},
issn = {2045-2322},
address = {[London]},
publisher = {Macmillan Publishers Limited, part of Springer Nature},
reportid = {DKFZ-2019-01929},
pages = {10924},
year = {2019},
abstract = {Keratins are the main cytoskeletal proteins of epithelial
cells and changes in the expression of keratins have
contributed to the evolutionary adaptation of epithelia to
different environments. Keratin K24 was proposed to be a
differentiation marker of epidermal keratinocytes but the
significance of K24 expression in the epidermis versus other
tissues has remained elusive. Here, we show by RT-PCR,
western blot, and immunofluorescence analyses that K24 is
highly expressed in the epithelium of the cornea whereas its
expression levels are significantly lower in other
stratified epithelia including in the epidermis. To
investigate the evolutionary history of K24, we screened the
genome sequences of vertebrates for orthologs of the human
KRT24 gene. The results of this comparative genomics study
suggested that KRT24 originated in a common ancestor of
amniotes and that it was lost independently in three clades
of mammals, i.e. camels, cetaceans, and a subclade of
pinnipeds comprising eared seals and the walrus. Together,
the results of this study identify K24 as component of the
cytoskeleton in the human corneal epithelium and reveal
previously unknown differences of keratin gene content among
mammalian species.},
cin = {A110},
ddc = {600},
cid = {I:(DE-He78)A110-20160331},
pnm = {311 - Signalling pathways, cell and tumor biology
(POF3-311)},
pid = {G:(DE-HGF)POF3-311},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:31358806},
pmc = {pmc:PMC6662840},
doi = {10.1038/s41598-019-47422-y},
url = {https://inrepo02.dkfz.de/record/144478},
}