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@ARTICLE{Vrieling:144880,
author = {A. Vrieling and H. B. Bueno-De-Mesquita and M. M. Ros and
E. Kampman and K. K. Aben and F. L. Büchner and E. H.
Jansen and N. Roswall and A. Tjønneland and M.-C.
Boutron-Ruault and C. Cadeau and J. Chang-Claude$^*$ and R.
Kaaks$^*$ and S. Weikert and H. Boeing and A. Trichopoulou
and P. Lagiou and D. Trichopoulos and S. Sieri and D. Palli
and S. Panico and P. H. Peeters and E. Weiderpass and G.
Skeie and P. Jakszyn and M.-D. Chirlaque and E. Ardanaz and
M.-J. Sánchez and R. Ehrnström and J. Malm and B.
Ljungberg and K.-T. Khaw and N. J. Wareham and P. Brennan
and M. Johansson and E. Riboli and L. A. Kiemeney},
title = {{O}ne-carbon metabolism biomarkers and risk of urothelial
cell carcinoma in the {E}uropean prospective investigation
into cancer and nutrition.},
journal = {International journal of cancer},
volume = {145},
number = {9},
issn = {1097-0215},
address = {Bognor Regis},
publisher = {Wiley-Liss},
reportid = {DKFZ-2019-02304},
pages = {2349 - 2359},
year = {2019},
abstract = {Published associations between dietary folate and bladder
cancer risk are inconsistent. Biomarkers may provide more
accurate measures of nutrient status. This nested
case-control analysis within the European Prospective
Investigation into Cancer and Nutrition (EPIC) investigated
associations between pre-diagnostic serum folate,
homocysteine, vitamins B6 and B12 and the risk of urothelial
cell carcinomas of the bladder (UCC). A total of 824
patients with newly diagnosed UCC were matched with 824
cohort members. Serum folate, homocysteine, and vitamins B6
and B12 were measured. Odds ratios (OR) and $95\%$
confidence intervals (CI) for total, aggressive, and
non-aggressive UCC were estimated using conditional logistic
regression with adjustment for smoking status, smoking
duration and intensity, and other potential confounders.
Additionally, statistical interaction with smoking status
was assessed. A halving in serum folate concentrations was
moderately associated with risk of UCC (OR: 1.18; $95\%$ CI:
0.98-1.43), in particular aggressive UCC (OR: 1.34; $95\%$
CI: 1.02-1.75; p-heterogeneity = 0.19). Compared to never
smokers in the highest quartile of folate concentrations,
this association seemed only apparent among current smokers
in the lowest quartile of folate concentrations (OR: 6.26;
$95\%$ CI: 3.62-10.81, p-interaction = 0.07). Dietary
folate was not associated with aggressive UCC (OR: 1.26;
$95\%$ CI: 0.81-1.95; p-heterogeneity = 0.14). No
association was observed between serum homocysteine,
vitamins B6 and B12 and risk of UCC. This study suggests
that lower serum folate concentrations are associated with
increased UCC risk, in particular aggressive UCC. Residual
confounding by smoking cannot be ruled out and these
findings require confirmation in future studies with
multiple measurements.},
cin = {C020},
ddc = {610},
cid = {I:(DE-He78)C020-20160331},
pnm = {313 - Cancer risk factors and prevention (POF3-313)},
pid = {G:(DE-HGF)POF3-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:30694528},
doi = {10.1002/ijc.32165},
url = {https://inrepo02.dkfz.de/record/144880},
}