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@ARTICLE{Paech:147181,
      author       = {D. Paech$^*$ and T. A. Kuder$^*$ and C. Roßmanith and M.
                      Griebe and P. Eisele and M. Platten and M. E. Ladd$^*$ and
                      H.-P. Schlemmer$^*$ and A. Gass and K. Szabo},
      title        = {{W}hat remains after transient global amnesia ({TGA})? {A}n
                      ultra-high field 7{T} magnetic resonance imaging study of
                      the hippocampus.},
      journal      = {European journal of neurology},
      volume       = {27},
      number       = {2},
      issn         = {1351-5101},
      address      = {Oxford},
      publisher    = {Blackwell Science65503},
      reportid     = {DKFZ-2019-02317},
      pages        = {406-409},
      year         = {2020},
      note         = {2020 Feb;27(2):406-409#EA:E010#},
      abstract     = {To study whether ultra-high-field 7T MRI can demonstrate
                      chronic focal defects in the hippocampus corresponding to
                      the former acute DWI lesions and to assess chronic
                      T2-hyperintense hippocampal lesion load in transient global
                      amnesia (TGA) patients.Follow-up 7T MRI of the hippocampus
                      was performed in 13 patients with documented hippocampal DWI
                      lesions (detected via 3T MRI) after acute TGA. The location
                      of DWI lesions was transformed to 7T T2 images after data
                      co-registration. Additionally, the T2-hyperintense lesion
                      load was estimated in each patient and compared with that of
                      13 healthy controls.7T MRI was performed after a median of
                      four months. No structural abnormality at the site of the
                      previous TGA lesion was observed in any case. None of the
                      controls showed DWI lesions. There was no significant
                      difference between patients and controls concerning the
                      number (p=0.67) or volume (p=0.45) of T2-hyperintense
                      hippocampal lesions.DWI lesions in patients with TGA do not
                      provoke any visible sequelae and do not result in
                      hippocampal cavities. The occurrence of incidental
                      hippocampal T2 lesions after TGA is not more frequent than
                      in controls.},
      cin          = {E010 / E020},
      ddc          = {610},
      cid          = {I:(DE-He78)E010-20160331 / I:(DE-He78)E020-20160331},
      pnm          = {315 - Imaging and radiooncology (POF3-315)},
      pid          = {G:(DE-HGF)POF3-315},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:31573112},
      doi          = {10.1111/ene.14099},
      url          = {https://inrepo02.dkfz.de/record/147181},
}