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@ARTICLE{Gao:147188,
author = {X. Gao$^*$ and Y. Xuan$^*$ and A. Benner$^*$ and A.
Anusruti$^*$ and H. Brenner$^*$ and B. Schöttker$^*$},
title = {{N}itric {O}xide {M}etabolites and {L}ung {C}ancer
{I}ncidence: {A} {M}atched {C}ase-{C}ontrol {S}tudy {N}ested
in the {ESTHER} {C}ohort.},
journal = {Oxidative medicine and cellular longevity},
volume = {2019},
issn = {1942-0994},
address = {Austin, Tex.},
publisher = {Landes Bioscience},
reportid = {DKFZ-2019-02324},
pages = {6470950},
year = {2019},
abstract = {Studies suggest that nitric oxide (NO) may have a possible
role in lung carcinogenesis. This study is aimed to evaluate
the association of the NO metabolites, namely, nitrite and
nitrate, with lung cancer incidence. We conducted a matched
case-control study (n = 245 incident lung cancer cases and n
= 735 controls) based on the German ESTHER cohort (n =
9,940). Controls were matched to cases on age, sex, smoking
status (never/former/current smoking), and pack-years of
smoking. The sum of nitrite and nitrate was measured in
urine samples using a colorimetric assay and was
standardized for renal function by urinary creatinine.
Conditional logistic regression models, adjusted for
lifestyle factors, asthma prevalence, and family history of
lung cancer, were used to estimate odds ratios (ORs) and
$95\%$ confidence intervals $(95\%$ CI). Among incident lung
cancer cases, high nitrite/nitrate levels were statistically
significantly associated with current smoking, a low BMI,
and the oxidative stress biomarker 8-isoprostane levels.
Nitrite/nitrate levels in the top quintile were
statistically significantly associated with lung cancer
incidence: the OR $(95\%$ CI) was 1.37 (1.04-1.82) for
comparison with the bottom quintile. This association was
unaltered after additional adjustment for 8-isoprostane
levels and C-reactive protein (CRP). In conclusion, this
large cohort study suggested that subjects with high urinary
nitrite/nitrate concentrations had an increased risk of lung
cancer and this association was independent of smoking, CRP,
8-isoprostane levels, and other established lung cancer risk
factors. Further studies are needed to validate these
findings and to confirm the hypothesis that pathologically
high levels of NO are involved in lung cancer development.},
cin = {C070 / C120 / C060 / L101},
ddc = {610},
cid = {I:(DE-He78)C070-20160331 / I:(DE-He78)C120-20160331 /
I:(DE-He78)C060-20160331 / I:(DE-He78)L101-20160331},
pnm = {313 - Cancer risk factors and prevention (POF3-313)},
pid = {G:(DE-HGF)POF3-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:31565153},
pmc = {pmc:PMC6745103},
doi = {10.1155/2019/6470950},
url = {https://inrepo02.dkfz.de/record/147188},
}