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@ARTICLE{Sasamoto:148282,
author = {N. Sasamoto and A. Babic and B. A. Rosner and R. T.
Fortner$^*$ and A. F. Vitonis and H. Yamamoto and R. N.
Fichorova and L. J. Titus and A. Tjønneland and L. Hansen
and M. Kvaskoff and A. Fournier and F. R. Mancini and H.
Boeing and A. Trichopoulou and E. Peppa and A. Karakatsani
and D. Palli and S. Grioni and A. Mattiello and R. Tumino
and V. Fiano and N. C. Onland-Moret and E. Weiderpass and I.
T. Gram and J. R. Quirós and L. Lujan-Barroso and M.-J.
Sánchez and S. Colorado-Yohar and A. Barricarte and P.
Amiano and A. Idahl and E. Lundin and H. Sartor and K.-T.
Khaw and T. J. Key and D. Muller and E. Riboli and M. Gunter
and L. Dossus and B. Trabert and N. Wentzensen and R.
Kaaks$^*$ and D. W. Cramer and S. S. Tworoger and K. L.
Terry},
title = {{D}evelopment and validation of circulating {CA}125
prediction models in postmenopausal women.},
journal = {Journal of ovarian research},
volume = {12},
number = {1},
issn = {1757-2215},
address = {London},
publisher = {BioMed Central},
reportid = {DKFZ-2019-02848},
pages = {116},
year = {2019},
abstract = {Cancer Antigen 125 (CA125) is currently the best available
ovarian cancer screening biomarker. However, CA125 has been
limited by low sensitivity and specificity in part due to
normal variation between individuals. Personal
characteristics that influence CA125 could be used to
improve its performance as screening biomarker.We developed
and validated linear and dichotomous (≥35 U/mL)
circulating CA125 prediction models in postmenopausal women
without ovarian cancer who participated in one of five large
population-based studies: Prostate, Lung, Colorectal, and
Ovarian Cancer Screening Trial (PLCO, n = 26,981),
European Prospective Investigation into Cancer and Nutrition
(EPIC, n = 861), the Nurses' Health Studies (NHS/NHSII,
n = 81), and the New England Case Control Study (NEC,
n = 923). The prediction models were developed using
stepwise regression in PLCO and validated in EPIC, NHS/NHSII
and NEC.The linear CA125 prediction model, which included
age, race, body mass index (BMI), smoking status and
duration, parity, hysterectomy, age at menopause, and
duration of hormone therapy (HT), explained $5\%$ of the
total variance of CA125. The correlation between measured
and predicted CA125 was comparable in PLCO testing dataset
(r = 0.18) and external validation datasets
(r = 0.14). The dichotomous CA125 prediction model
included age, race, BMI, smoking status and duration,
hysterectomy, time since menopause, and duration of HT with
AUC of 0.64 in PLCO and 0.80 in validation dataset.The
linear prediction model explained a small portion of the
total variability of CA125, suggesting the need to identify
novel predictors of CA125. The dichotomous prediction model
showed moderate discriminatory performance which validated
well in independent dataset. Our dichotomous model could be
valuable in identifying healthy women who may have elevated
CA125 levels, which may contribute to reducing false
positive tests using CA125 as screening biomarker.},
cin = {C020},
ddc = {610},
cid = {I:(DE-He78)C020-20160331},
pnm = {313 - Cancer risk factors and prevention (POF3-313)},
pid = {G:(DE-HGF)POF3-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:31771659},
pmc = {pmc:PMC6878636},
doi = {10.1186/s13048-019-0591-4},
url = {https://inrepo02.dkfz.de/record/148282},
}