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@ARTICLE{Singhal:148771,
author = {M. Singhal$^*$ and H. Augustin$^*$},
title = {{B}eyond angiogenesis: {E}xploiting angiocrine factors to
restrict tumor progression and metastasis.},
journal = {Cancer research},
volume = {80},
number = {4},
issn = {1538-7445},
address = {Philadelphia, Pa.},
publisher = {AACR},
reportid = {DKFZ-2019-03284},
pages = {659-662},
year = {2020},
note = {DKFZ-ZMBH Alliance 2020 Feb
15;80(4):659-662#EA:A190#LA:A190#},
abstract = {Looking beyond tumor angiogenesis, the past decade has
witnessed a fundamental change of paradigm with the
discovery that the vascular endothelium does not just
respond to exogenous cytokines, but exerts active
'angiocrine' gatekeeper roles controling their
microenvironment in an instructive manner. While vascular
niches host disseminated cancer cells and promote their
stemness, endothelial cell-derived angiocrine signals
orchestrate a favorable immune milieu to facilitate
metastatic growth. Here, we discuss recent advances in the
field of tumor microenvironment research and propose
angiocrine signals as promising targets of future
mechanism-driven anti-metastatic therapies, which may prove
useful to synergistically combine with chemotherapy and
immunotherapy.},
cin = {A190 / HD01},
ddc = {610},
cid = {I:(DE-He78)A190-20160331 / I:(DE-He78)HD01-20160331},
pnm = {311 - Signalling pathways, cell and tumor biology
(POF3-311)},
pid = {G:(DE-HGF)POF3-311},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:31831463},
doi = {10.1158/0008-5472.CAN-19-3351},
url = {https://inrepo02.dkfz.de/record/148771},
}