000153601 001__ 153601 000153601 005__ 20240320151820.0 000153601 0247_ $$2doi$$a10.1038/s41586-019-1907-7 000153601 0247_ $$2pmid$$apmid:32025013 000153601 0247_ $$2ISSN$$a0028-0836 000153601 0247_ $$2ISSN$$a1476-4687 000153601 0247_ $$2altmetric$$aaltmetric:75075728 000153601 037__ $$aDKFZ-2020-00345 000153601 041__ $$aeng 000153601 082__ $$a500 000153601 1001_ $$aGerstung, Moritz$$b0 000153601 245__ $$aThe evolutionary history of 2,658 cancers. 000153601 260__ $$aLondon [u.a.]$$bNature Publ. Group52462$$c2020 000153601 3367_ $$2DRIVER$$aarticle 000153601 3367_ $$2DataCite$$aOutput Types/Journal article 000153601 3367_ $$0PUB:(DE-HGF)16$$2PUB:(DE-HGF)$$aJournal Article$$bjournal$$mjournal$$s1710944072_4145 000153601 3367_ $$2BibTeX$$aARTICLE 000153601 3367_ $$2ORCID$$aJOURNAL_ARTICLE 000153601 3367_ $$00$$2EndNote$$aJournal Article 000153601 500__ $$asiehe Correction: DKFZ Autoren affiliiert im PCAWG Consortium: https://inrepo02.dkfz.de/record/212474 / https://doi.org/10.1038/s41586-022-05601-4 000153601 520__ $$aCancer develops through a process of somatic evolution1,2. Sequencing data from a single biopsy represent a snapshot of this process that can reveal the timing of specific genomic aberrations and the changing influence of mutational processes3. Here, by whole-genome sequencing analysis of 2,658 cancers as part of the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA)4, we reconstruct the life history and evolution of mutational processes and driver mutation sequences of 38 types of cancer. Early oncogenesis is characterized by mutations in a constrained set of driver genes, and specific copy number gains, such as trisomy 7 in glioblastoma and isochromosome 17q in medulloblastoma. The mutational spectrum changes significantly throughout tumour evolution in 40% of samples. A nearly fourfold diversification of driver genes and increased genomic instability are features of later stages. Copy number alterations often occur in mitotic crises, and lead to simultaneous gains of chromosomal segments. Timing analyses suggest that driver mutations often precede diagnosis by many years, if not decades. Together, these results determine the evolutionary trajectories of cancer, and highlight opportunities for early cancer detection. 000153601 536__ $$0G:(DE-HGF)POF3-312$$a312 - Functional and structural genomics (POF3-312)$$cPOF3-312$$fPOF III$$x0 000153601 588__ $$aDataset connected to CrossRef, PubMed, 000153601 7001_ $$aJolly, Clemency$$b1 000153601 7001_ $$aLeshchiner, Ignaty$$b2 000153601 7001_ $$aDentro, Stefan$$b3 000153601 7001_ $$aGonzalez, Santiago$$b4 000153601 7001_ $$aRosebrock, Daniel$$b5 000153601 7001_ $$aMitchell, Thomas J$$b6 000153601 7001_ $$aRubanova, Yulia$$b7 000153601 7001_ $$aAnur, Pavana$$b8 000153601 7001_ $$aYu, Kaixian$$b9 000153601 7001_ $$aTarabichi, Maxime$$b10 000153601 7001_ $$aDeshwar, Amit$$b11 000153601 7001_ $$aWintersinger, Jeff$$b12 000153601 7001_ $$0P:(DE-He78)e053817bc90becc8a8dd8250e677e773$$aKleinheinz, Kortine$$b13 000153601 7001_ $$aVázquez-García, Ignacio$$b14 000153601 7001_ $$aHaase, Kerstin$$b15 000153601 7001_ $$aJerman, Lara$$b16 000153601 7001_ $$aSengupta, Subhajit$$b17 000153601 7001_ $$aMacintyre, Geoff$$b18 000153601 7001_ $$aMalikic, Salem$$b19 000153601 7001_ $$aDonmez, Nilgun$$b20 000153601 7001_ $$aLivitz, Dimitri G$$b21 000153601 7001_ $$aCmero, Marek$$b22 000153601 7001_ $$aDemeulemeester, Jonas$$b23 000153601 7001_ $$aSchumacher, Steven$$b24 000153601 7001_ $$aFan, Yu$$b25 000153601 7001_ $$aYao, Xiaotong$$b26 000153601 7001_ $$aLee, Juhee$$b27 000153601 7001_ $$0P:(DE-He78)f2a782242acf94a3114d75c45dc75b37$$aSchlesner, Matthias$$b28 000153601 7001_ $$aBoutros, Paul C$$b29 000153601 7001_ $$aBowtell, David D$$b30 000153601 7001_ $$aZhu, Hongtu$$b31 000153601 7001_ $$aGetz, Gad$$b32 000153601 7001_ $$aImielinski, Marcin$$b33 000153601 7001_ $$aBeroukhim, Rameen$$b34 000153601 7001_ $$aSahinalp, S Cenk$$b35 000153601 7001_ $$aJi, Yuan$$b36 000153601 7001_ $$aPeifer, Martin$$b37 000153601 7001_ $$aMarkowetz, Florian$$b38 000153601 7001_ $$aMustonen, Ville$$b39 000153601 7001_ $$aYuan, Ke$$b40 000153601 7001_ $$aWang, Wenyi$$b41 000153601 7001_ $$aMorris, Quaid D$$b42 000153601 7001_ $$0P:(DE-HGF)0$$aPCAWGEvolution&HeterogeneityWorkingGroup$$b43 000153601 7001_ $$aSpellman, Paul T$$b44 000153601 7001_ $$aWedge, David C$$b45 000153601 7001_ $$aVan Loo, Peter$$b46 000153601 7001_ $$0P:(DE-HGF)0$$aPCAWGConsortium$$b47 000153601 773__ $$0PERI:(DE-600)1413423-8$$a10.1038/s41586-019-1907-7$$gVol. 578, no. 7793, p. 122 - 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