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@ARTICLE{Sowah:153725,
author = {S. Sowah$^*$ and F. Hirche and A. Milanese and T. S.
Johnson$^*$ and M. Grafetstätter$^*$ and R. Schübel$^*$
and R. Kirsten and C. M. Ulrich and R. Kaaks$^*$ and G.
Zeller and T. Kühn$^*$ and G. I. Stangl},
title = {{C}hanges in {P}lasma {S}hort-{C}hain {F}atty {A}cid
{L}evels after {D}ietary {W}eight {L}oss {A}mong
{O}verweight and {O}bese {A}dults over 50 {W}eeks.},
journal = {Nutrients},
volume = {12},
number = {2},
issn = {2072-6643},
address = {Basel},
publisher = {MDPI},
reportid = {DKFZ-2020-00422},
pages = {452},
year = {2020},
note = {#EA:C020#},
abstract = {Gut microbial-derived short-chain fatty acids (SCFAs) may
regulate energy homeostasis and exert anti-carcinogenic,
immunomodulatory and anti-inflammatory effects. Smaller
trials indicate that dietary weight loss may lead to
decreased SCFA production, but findings have been
inconclusive. SCFA concentrations were measured by
HPLC-MS/MS in plasma samples of 150 overweight or obese
adults in a trial initially designed to evaluate the
metabolic effects of intermittent (ICR) versus continuous
(CCR) calorie restriction (NCT02449148). For the present
post hoc analyses, participants were classified by quartiles
of weight loss, irrespective of the dietary intervention.
Linear mixed models were used to analyze weight-loss-induced
changes in SCFA concentrations after 12, 24 and 50 weeks.
There were no differential changes in SCFA levels across the
initial study arms (ICR versus CCR versus control) after 12
weeks, but acetate concentrations significantly decreased
with overall weight loss (mean log-relative change of -0.7
± 1.8 in the lowest quartile versus. -7.6 ± 2 in the
highest, p = 0.026). Concentrations of propionate, butyrate
and other SCFAs did not change throughout the study. Our
results show that weight-loss, achieved through calorie
restriction, may lead to smaller initial decreases in plasma
acetate, while plasma SCFAs generally remain remarkably
stable over time.},
cin = {C020},
ddc = {610},
cid = {I:(DE-He78)C020-20160331},
pnm = {313 - Cancer risk factors and prevention (POF3-313)},
pid = {G:(DE-HGF)POF3-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:32053988},
doi = {10.3390/nu12020452},
url = {https://inrepo02.dkfz.de/record/153725},
}