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@ARTICLE{Wierzbinska:154170,
author = {J. A. Wierzbinska$^*$ and R. Toth$^*$ and N. Ishaque$^*$
and K. Rippe$^*$ and J.-P. Mallm$^*$ and L. C. Klett$^*$ and
D. Mertens$^*$ and T. Zenz and T. Hielscher$^*$ and M.
Seifert and R. Küppers and Y. Assenov$^*$ and P. Lutsik$^*$
and S. Stilgenbauer and P. M. Roessner$^*$ and M.
Seiffert$^*$ and J. Byrd and C. C. Oakes and C. Plass$^*$
and D. Lipka$^*$},
title = {{M}ethylome-based cell-of-origin modeling ({M}ethyl-{COOM})
identifies aberrant expression of immune regulatory
molecules in {CLL}.},
journal = {Genome medicine},
volume = {12},
number = {1},
issn = {1756-994X},
address = {London},
publisher = {BioMed Central},
reportid = {DKFZ-2020-00629},
pages = {29},
year = {2020},
note = {Modyfied by adding this comment for testin
purposes#EA:B340#LA:B340#},
abstract = {In cancer, normal epigenetic patterns are disturbed and
contribute to gene expression changes, disease onset, and
progression. The cancer epigenome is composed of the
epigenetic patterns present in the tumor-initiating cell at
the time of transformation, and the tumor-specific
epigenetic alterations that are acquired during tumor
initiation and progression. The precise dissection of these
two components of the tumor epigenome will facilitate a
better understanding of the biological mechanisms underlying
malignant transformation. Chronic lymphocytic leukemia (CLL)
originates from differentiating B cells, which undergo
extensive epigenetic programming. This poses the challenge
to precisely determine the epigenomic ground state of the
cell-of-origin in order to identify CLL-specific epigenetic
aberrations.We developed a linear regression model,
methylome-based cell-of-origin modeling (Methyl-COOM), to
map the cell-of-origin for individual CLL patients based on
the continuum of epigenomic changes during normal B cell
differentiation.Methyl-COOM accurately maps the
cell-of-origin of CLL and identifies CLL-specific aberrant
DNA methylation events that are not confounded by
physiologic epigenetic B cell programming. Furthermore,
Methyl-COOM unmasks abnormal action of transcription
factors, altered super-enhancer activities, and aberrant
transcript expression in CLL. Among the aberrantly regulated
transcripts were many genes that have previously been
implicated in T cell biology. Flow cytometry analysis of
these markers confirmed their aberrant expression on
malignant B cells at the protein level.Methyl-COOM analysis
of CLL identified disease-specific aberrant gene regulation.
The aberrantly expressed genes identified in this study
might play a role in immune-evasion in CLL and might serve
as novel targets for immunotherapy approaches. In summary,
we propose a novel framework for in silico modeling of
reference DNA methylomes and for the identification of
cancer-specific epigenetic changes, a concept that can be
broadly applied to other human malignancies.},
cin = {B370 / HD01 / B066 / B061 / C060 / B060 / B340},
ddc = {610},
cid = {I:(DE-He78)B370-20160331 / I:(DE-He78)HD01-20160331 /
I:(DE-He78)B066-20160331 / I:(DE-He78)B061-20160331 /
I:(DE-He78)C060-20160331 / I:(DE-He78)B060-20160331 /
I:(DE-He78)B340-20160331},
pnm = {312 - Functional and structural genomics (POF3-312)},
pid = {G:(DE-HGF)POF3-312},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:32188505},
doi = {10.1186/s13073-020-00724-7},
url = {https://inrepo02.dkfz.de/record/154170},
}