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@ARTICLE{Peters:154696,
      author       = {S. Peters and V. Gallo and P. Vineis and L. T. Middleton
                      and L. Forsgren and C. Sacerdote and S. Sieri and A. Kyrozis
                      and M.-D. Chirlaque and R. Zamora-Ros and O. Hansson and J.
                      Petersson and V. Katzke$^*$ and T. Kühn$^*$ and O. Mokoroa
                      and G. Masala and E. Ardanaz and S. Panico and M. M.
                      Bergmann and T. J. Key and E. Weiderpass and P. Ferrari and
                      R. Vermeulen},
      title        = {{A}lcohol {C}onsumption and {R}isk of {P}arkinson's
                      {D}isease: {D}ata from a {L}arge {P}rospective {E}uropean
                      {C}ohort.},
      journal      = {Movement disorders},
      volume       = {35},
      number       = {7},
      issn         = {1531-8257},
      address      = {New York, NY},
      publisher    = {Wiley},
      reportid     = {DKFZ-2020-00954},
      pages        = {1258-1263},
      year         = {2020},
      note         = {2020 Jul;35(7):1258-1263},
      abstract     = {Parkinson's disease (PD) etiology is not well understood.
                      Reported inverse associations with smoking and coffee
                      consumption prompted the investigation of alcohol
                      consumption as a risk factor, for which evidence is
                      inconclusive.To assess the associations between alcohol
                      consumption and PD risk.Within NeuroEPIC4PD, a prospective
                      European population-based cohort, 694 incident PD cases were
                      ascertained from 209,998 PD-free participants. Average
                      alcohol consumption at different time points was
                      self-reported at recruitment. Cox regression hazard ratios
                      were estimated for alcohol consumption and PD occurrence.No
                      associations between baseline or lifetime total alcohol
                      consumption and PD risk were observed. Men with moderate
                      lifetime consumption (5-29.9 g/day) were at $~50\%$ higher
                      risk compared with light consumption (0.1-4.9 g/day), but no
                      linear exposure-response trend was observed. Analyses by
                      beverage type also revealed no associations with PD.Our data
                      reinforce previous findings from prospective studies showing
                      no association between alcohol consumption and PD risk. ©
                      2020 The Authors. Movement Disorders published by Wiley
                      Periodicals, Inc. on behalf of International Parkinson and
                      Movement Disorder Society.},
      cin          = {C020},
      ddc          = {610},
      cid          = {I:(DE-He78)C020-20160331},
      pnm          = {313 - Cancer risk factors and prevention (POF3-313)},
      pid          = {G:(DE-HGF)POF3-313},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:32357270},
      doi          = {10.1002/mds.28039},
      url          = {https://inrepo02.dkfz.de/record/154696},
}