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@ARTICLE{Gengenbacher:164197,
author = {N. Gengenbacher$^*$ and M. Singhal$^*$ and C. Mogler and L.
Hai$^*$ and L. Milde$^*$ and A. A. Abdul Pari$^*$ and E.
Besemfelder$^*$ and C. Fricke$^*$ and D. Baumann$^*$ and S.
Gehrs$^*$ and J. Utikal$^*$ and M. Felcht and J. Hu and M.
Schlesner$^*$ and R. Offringa$^*$ and S. Chintharlapalli and
H. Augustin$^*$},
title = {{T}imed {A}ng2-targeted therapy identifies the
{A}ngiopoietin-{T}ie pathway as key regulator of fatal
lymphogenous metastasis.},
journal = {Cancer discovery},
volume = {11},
number = {2},
issn = {2159-8290},
address = {Philadelphia, Pa.},
reportid = {DKFZ-2020-02307},
pages = {424-445},
year = {2021},
note = {DKFZ-ZMBH Alliance#EA:A190#LA:A190#2021 Feb;11(2):424-445},
abstract = {Recent clinical and preclinical advances have highlighted
the existence of a previously hypothesized lymphogenous
route of metastasis. However, due to a lack of suitable
preclinical modeling tools, its contribution to long-term
disease outcome and relevance for therapy remain
controversial. Here, we established a GEMM fragment-based
tumor model uniquely sustaining a functional network of
intratumoral lymphatics that facilitates seeding of fatal
peripheral metastases. Multi-regimen survival studies and
correlative patient data identified primary tumor-derived
Angiopoietin-2 (Ang2) as a potent therapeutic target to
restrict lymphogenous tumor cell dissemination.
Mechanistically, tumor-associated lymphatic endothelial
cells (EC), in contrast to blood vascular EC, were found to
be critically addicted to the Angiopoietin-Tie pathway.
Genetic manipulation experiments in combination with
single-cell mapping revealed agonistically-acting
Ang2/Tie2-signaling as key regulator of lymphatic
maintenance. Correspondingly, acute pre-surgical
Ang2-neutralization was sufficient to prolong survival by
regressing established intratumoral lymphatics, hence
identifying a novel therapeutic regimen that warrants
further clinical evaluation.},
cin = {A190 / B240 / D200 / A370},
ddc = {610},
cid = {I:(DE-He78)A190-20160331 / I:(DE-He78)B240-20160331 /
I:(DE-He78)D200-20160331 / I:(DE-He78)A370-20160331},
pnm = {311 - Zellbiologie und Tumorbiologie (POF4-311)},
pid = {G:(DE-HGF)POF4-311},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:33106316},
doi = {10.1158/2159-8290.CD-20-0122},
url = {https://inrepo02.dkfz.de/record/164197},
}