TY - JOUR
AU - Brandão, Andreia
AU - Paulo, Paula
AU - Maia, Sofia
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AU - Peixoto, Ana
AU - Cardoso, Marta
AU - Silva, Maria P
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AU - Neal, David E
AU - Nordestgaard, Børge G
AU - Tangen, Catherine M
AU - Southey, Melissa C
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AU - Albanes, Demetrius
AU - Haiman, Christopher A
AU - Travis, Ruth C
AU - Stanford, Janet L
AU - Mucci, Lorelei A
AU - West, Catharine M L
AU - Nielsen, Sune F
AU - Kibel, Adam S
AU - Cussenot, Olivier
AU - Berndt, Sonja I
AU - Koutros, Stella
AU - Sørensen, Karina Dalsgaard
AU - Cybulski, Cezary
AU - Grindedal, Eli Marie
AU - Park, Jong Y
AU - Ingles, Sue A
AU - Maier, Christiane
AU - Hamilton, Robert J
AU - Rosenstein, Barry S
AU - Vega, Ana
AU - The Impact Study Steering Committee And Collaborators
AU - Kogevinas, Manolis
AU - Wiklund, Fredrik
AU - Penney, Kathryn L
AU - Brenner, Hermann
AU - John, Esther M
AU - Kaneva, Radka
AU - Logothetis, Christopher J
AU - Neuhausen, Susan L
AU - Ruyck, Kim De
AU - Razack, Azad
AU - Newcomb, Lisa F
AU - Canary Pass Investigators
AU - Lessel, Davor
AU - Usmani, Nawaid
AU - Claessens, Frank
AU - Gago-Dominguez, Manuela
AU - Townsend, Paul A
AU - Roobol, Monique J
AU - The Profile Study Steering Committee
AU - The Practical Consortium
AU - Teixeira, Manuel R
TI - The CHEK2 Variant C.349A</td><td width="150">
TI - gt;G Is Associated with Prostate Cancer Risk and Carriers Share a Common Ancestor.
JO - Cancers
VL - 12
IS - 11
SN - 2072-6694
CY - Basel
PB - MDPI
M1 - DKFZ-2020-02405
SP - 3254
PY - 2020
AB - The identification of recurrent founder variants in cancer predisposing genes may have important implications for implementing cost-effective targeted genetic screening strategies. In this study, we evaluated the prevalence and relative risk of the CHEK2 recurrent variant c.349A>G in a series of 462 Portuguese patients with early-onset and/or familial/hereditary prostate cancer (PrCa), as well as in the large multicentre PRACTICAL case-control study comprising 55,162 prostate cancer cases and 36,147 controls. Additionally, we investigated the potential shared ancestry of the carriers by performing identity-by-descent, haplotype and age estimation analyses using high-density SNP data from 70 variant carriers belonging to 11 different populations included in the PRACTICAL consortium. The CHEK2 missense variant c.349A>G was found significantly associated with an increased risk for PrCa (OR 1.9; 95
LB - PUB:(DE-HGF)16
C6 - pmid:33158149
DO - DOI:10.3390/cancers12113254
UR - https://inrepo02.dkfz.de/record/165825
ER -