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@ARTICLE{Thomsen:166140,
      author       = {H. Thomsen$^*$ and X. Li and K. Sundquist and J. Sundquist
                      and A. Försti$^*$ and K. Hemminki$^*$},
      title        = {{F}amilial risks between giant cell arteritis and
                      {T}akayasu arteritis and other autoimmune diseases in the
                      population of {S}weden.},
      journal      = {Scientific reports},
      volume       = {10},
      number       = {1},
      issn         = {2045-2322},
      address      = {[London]},
      publisher    = {Macmillan Publishers Limited, part of Springer Nature},
      reportid     = {DKFZ-2020-02648},
      pages        = {20887},
      year         = {2020},
      note         = {#EA:C050#LA:C050#LA:C020#},
      abstract     = {Giant cell arteritis (GCA, also called temporal arteritis)
                      is a rare and Takayasu arteritis (TA) is an even rarer
                      autoimmune disease (AID), both of which present with
                      inflammatory vasculitis of large and medium size arteries.
                      The risk factors are largely undefined but disease
                      susceptibility has been associated with human leukocyte
                      antigen locus. Population-level familial risk is not known.
                      In the present nation-wide study we describe familial risk
                      for GCA and for GCA and TA with any other AID based on the
                      Swedish hospital diagnoses up to years 2012. Family
                      relationships were obtained from the Multigeneration
                      Register. Familial standardized incidence ratios (SIRs) were
                      calculated for offspring whose parents or siblings were
                      diagnosed with GCA, TA or any other AID. The number of GCA
                      patients in the offspring generation was 4695, compared to
                      209 TA patients; for both, familial patients accounted for
                      $1\%$ of all patients. The familial risk for GCA was 2.14,
                      2.40 for women and non-significant for men. GCA was
                      associated with 10 other AIDs and TA was associated with 6
                      other AIDs; both shared associations with polymyalgia
                      rheumatica and rheumatoid arthritis. The results showed that
                      family history is a risk factor for GCA. Significant
                      familial associations of both GCA and TA with such a number
                      of other AIDs provide evidence for polyautoimmunity among
                      these diseases.},
      cin          = {C050 / HD01 / B062 / C020},
      ddc          = {600},
      cid          = {I:(DE-He78)C050-20160331 / I:(DE-He78)HD01-20160331 /
                      I:(DE-He78)B062-20160331 / I:(DE-He78)C020-20160331},
      pnm          = {313 - Cancer risk factors and prevention (POF3-313)},
      pid          = {G:(DE-HGF)POF3-313},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:33257751},
      doi          = {10.1038/s41598-020-77857-7},
      url          = {https://inrepo02.dkfz.de/record/166140},
}