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@MASTERSTHESIS{Camazzola:166145,
author = {G. Camazzola$^*$},
title = {{M}odulation of intracellular oxygen:making the cell
nucleus hypoxic forextracellular oxic conditions},
school = {Universität Heidelberg},
type = {Masterarbeit},
reportid = {DKFZ-2020-02653},
year = {2020},
note = {Masterarbeit, Universität Heidelberg, 2020},
abstract = {In the presented work the analysis of a novel molecular
mechanism is proposed, by whichcells can modulate their
intracellular oxygen and potentially become hypoxic,
inducingfavourable circumstances for metabolic
reprogramming, even under extracellular oxic conditions.
This phenomenon is based on the competition between O2
diffusion, from theextracellular environment to the nucleus,
and its consumption, especially by NOX enzymes.The novelty
revolves around the assumptions that the endoplasmic
reticulum (ER) membranes can lower the diffusion rate of
oxygen, and that the sensing enzymes become moreeffective at
removing O2for low values of the Michaelis constant KM.
After a preliminarystudy of oxygen concentration inside the
cell, an analysis of different factors affecting
thisphenomenon was carried out. Attention was given to the
Michaelis-Menten parametersVmax and KM, considering
consumption differences between ER and cytosol, the
extracellular oxygen, the number of membrane pairs, the
membrane porosity and the dependence onthe cell cycle phase.
A relation between porosity and number of membranes was
established,and the ratio of nuclear O2 of G1 phase relative
to S phase, for different KM, was determined. In conclusion,
the initial hypothesis has been confirmed, and some
experimentalresults obtained in previous studies have been
validated.},
cin = {E041},
cid = {I:(DE-He78)E041-20160331},
pnm = {315 - Imaging and radiooncology (POF3-315)},
pid = {G:(DE-HGF)POF3-315},
typ = {PUB:(DE-HGF)19},
url = {https://inrepo02.dkfz.de/record/166145},
}