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@ARTICLE{Paredes:167282,
author = {I. Paredes and J. R. Vieira and B. Shah and C. F. Ramunno
and J. Dyckow and H. Adler and M. Richter and G. Schermann
and E. Giannakouri$^*$ and L. Schirmer and H. G.
Augustin$^*$ and C. Ruiz de Almodóvar},
title = {{O}ligodendrocyte precursor cell specification is regulated
by bidirectional neural progenitor-endothelial cell
crosstalk.},
journal = {Nature neuroscience},
volume = {24},
number = {4},
issn = {1546-1726},
address = {New York, NY},
publisher = {Nature America},
reportid = {DKFZ-2021-00237},
pages = {478-488},
year = {2021},
note = {DKFZ-ZMBH Alliance / 2021 Apr;24(4):478-488},
abstract = {Neural-derived signals are crucial regulators of CNS
vascularization. However, whether the vasculature responds
to these signals by means of elongating and branching or in
addition by building a feedback response to modulate
neurodevelopmental processes remains unknown. In this study,
we identified bidirectional crosstalk between the neural and
the vascular compartment of the developing CNS required for
oligodendrocyte precursor cell specification.
Mechanistically, we show that neural progenitor cells (NPCs)
express angiopoietin-1 (Ang1) and that this expression is
regulated by Sonic hedgehog. We demonstrate that NPC-derived
Ang1 signals to its receptor, Tie2, on endothelial cells to
induce the production of transforming growth factor beta 1
(TGFβ1). Endothelial-derived TGFβ1, in turn, acts as an
angiocrine molecule and signals back to NPCs to induce their
commitment toward oligodendrocyte precursor cells. This work
demonstrates a true bidirectional collaboration between NPCs
and the vasculature as a critical regulator of
oligodendrogenesis.},
cin = {A190},
ddc = {610},
cid = {I:(DE-He78)A190-20160331},
pnm = {311 - Zellbiologie und Tumorbiologie (POF4-311)},
pid = {G:(DE-HGF)POF4-311},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:33510480},
doi = {10.1038/s41593-020-00788-z},
url = {https://inrepo02.dkfz.de/record/167282},
}