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@ARTICLE{Welzel:168288,
author = {T. Welzel and A. Bendinger$^*$ and C. Glowa$^*$ and I.
Babushkina$^*$ and M. Jugold$^*$ and P. Peschke$^*$ and J.
Debus$^*$ and C. P. Karger$^*$ and M. Saager$^*$},
title = {{L}ongitudinal {MRI} study after carbon ion and photon
irradiation: shorter latency time for myelopathy is not
associated with differential morphological changes.},
journal = {Radiation oncology},
volume = {16},
number = {1},
issn = {1748-717X},
address = {London},
publisher = {BioMed Central},
reportid = {DKFZ-2021-00799},
pages = {63},
year = {2021},
note = {#EA:E040#LA:E040#},
abstract = {Radiation-induced myelopathy is a severe and irreversible
complication that occurs after a long symptom-free latency
time if the spinal cord was exposed to a significant
irradiation dose during tumor treatment. As carbon ions are
increasingly investigated for tumor treatment in clinical
trials, their effect on normal tissue needs further
investigation to assure safety of patient treatments.
Magnetic resonance imaging (MRI)-visible morphological
alterations could serve as predictive markers for medicinal
interventions to avoid severe side effects. Thus,
MRI-visible morphological alterations in the rat spinal cord
after high dose photon and carbon ion irradiation and their
latency times were investigated.Rats whose spinal cords were
irradiated with iso-effective high photon (n = 8) or carbon
ion (n = 8) doses as well as sham-treated control animals (n
= 6) underwent frequent MRI measurements until they
developed radiation-induced myelopathy (paresis II). MR
images were analyzed for morphological alterations and
animals were regularly tested for neurological deficits. In
addition, histological analysis was performed of animals
suffering from paresis II compared to controls.For both beam
modalities, first morphological alterations occurred outside
the spinal cord (bone marrow conversion, contrast agent
accumulation in the musculature ventral and dorsal to the
spinal cord) followed by morphological alterations inside
the spinal cord (edema, syrinx, contrast agent accumulation)
and eventually neurological alterations (paresis I and II).
Latency times were significantly shorter after carbon ions
as compared to photon irradiation.Irradiation of the rat
spinal cord with photon or carbon ion doses that lead to
$100\%$ myelopathy induced a comparable fixed sequence of
MRI-visible morphological alterations and neurological
distortions. However, at least in the animal model used in
this study, the observed MRI-visible morphological
alterations in the spinal cord are not suited as predictive
markers to identify animals that will develop myelopathy as
the time between MRI-visible alterations and the occurrence
of myelopathy is too short to intervene with protective or
mitigative drugs.},
keywords = {Carbon ion irradiation (Other) / Cervical spinal cord
(Other) / Late radiation effects (Other) / Magnetic
resonance imaging (Other) / Myelopathy (Other)},
cin = {E040 / W240 / E050},
ddc = {610},
cid = {I:(DE-He78)E040-20160331 / I:(DE-He78)W240-20160331 /
I:(DE-He78)E050-20160331},
pnm = {315 - Bildgebung und Radioonkologie (POF4-315)},
pid = {G:(DE-HGF)POF4-315},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:33789720},
pmc = {pmc:PMC8011205},
doi = {10.1186/s13014-021-01792-8},
url = {https://inrepo02.dkfz.de/record/168288},
}