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@ARTICLE{Wutschka:169894,
author = {J. Wutschka$^*$ and B. Kast$^*$ and M. Sator-Schmitt$^*$
and S. Appak-Baskoy$^*$ and J. Hess$^*$ and H.-P. Sinn and
P. Angel$^*$ and M. Schorpp-Kistner$^*$},
title = {{JUNB} suppresses distant metastasis by influencing the
initial metastatic stage.},
journal = {Clinical $\&$ experimental metastasis},
volume = {38},
number = {4},
issn = {1573-7276},
address = {Dordrecht},
publisher = {Springer Science + Business Media B.V.},
reportid = {DKFZ-2021-01628},
pages = {411-423},
year = {2021},
note = {DKFZ-ZMBH Alliance / #EA:A100#LA:A100#/ 2021
Aug;38(4):411-423},
abstract = {The complex interactions between cells of the tumor
microenvironment and cancer cells are considered a major
determinant of cancer progression and metastasis. Yet, our
understanding of the mechanisms of metastatic disease is not
sufficient to successfully treat patients with
advanced-stage cancer. JUNB is a member of the AP-1
transcription factor family shown to be frequently
deregulated in human cancer and associated with invasion and
metastasis. A strikingly high stromal JUNB expression in
human breast cancer samples prompted us to functionally
investigate the consequences of JUNB loss in cells of the
tumor microenvironment on cancer progression and metastasis
in mice. To adequately mimic the clinical situation, we
applied a syngeneic spontaneous breast cancer metastasis
model followed by primary tumor resection and identified
stromal JUNB as a potent suppressor of distant metastasis.
Comprehensive characterization of the JUNB-deficient tumor
microenvironment revealed a strong influx of myeloid cells
into primary breast tumors and lungs at early metastatic
stage. In these infiltrating neutrophils, BV8 and MMP9,
proteins promoting angiogenesis and tissue remodeling, were
specifically upregulated in a JUNB-dependent manner. Taken
together, we established stromal JUNB as a strong suppressor
of distant metastasis. Consequently, therapeutic strategies
targeting AP-1 should be carefully designed not to interfere
with stromal JUNB expression as this may be detrimental for
cancer patients.},
keywords = {Metastasis (Other) / Transcription factor (Other) / Tumor
microenvironment (Other)},
cin = {A100 / E221},
ddc = {610},
cid = {I:(DE-He78)A100-20160331 / I:(DE-He78)E221-20160331},
pnm = {311 - Zellbiologie und Tumorbiologie (POF4-311)},
pid = {G:(DE-HGF)POF4-311},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:34282521},
doi = {10.1007/s10585-021-10108-9},
url = {https://inrepo02.dkfz.de/record/169894},
}