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@ARTICLE{OConnor:169932,
author = {T. L. O'Connor$^*$ and M. Heikenwälder$^*$},
title = {{CCL}2 in the {T}umor {M}icroenvironment.},
journal = {Advances in experimental medicine and biology},
volume = {1302},
issn = {0065-2598},
address = {[Heidelberg]},
publisher = {Springer},
reportid = {DKFZ-2021-01638},
isbn = {978-3-030-62657-0 (print)},
pages = {1-14},
year = {2021},
note = {#EA:F180#LA:F180#},
abstract = {The C-C motif chemokine ligand 2 (CCL2) is a crucial
mediator of immune cell recruitment during microbial
infections and tissue damage. CCL2 is also frequently
overexpressed in cancer cells and other cells in the tumor
microenvironment, and a large body of evidence indicates
that high CCL2 levels are associated with more aggressive
malignancies, a higher probability of metastasis, and poorer
outcomes in a wide range of cancers. CCL2 plays a role in
recruiting tumor-associated macrophages (TAMs), which adopt
a pro-tumorigenic phenotype and support cancer cell
survival, facilitate tumor cell invasion, and promote
angiogenesis. CCL2 also has direct, TAM-independent effects
on tumor cells and the tumor microenvironment, including
recruitment of other myeloid subsets and non-myeloid cells,
maintaining an immunosuppressive environment, stimulating
tumor cell growth and motility, and promoting angiogenesis.
CCL2 also plays important roles in the metastatic cascade,
such as creating a pre-metastatic niche in distant organs
and promoting tumor cell extravasation across endothelia.
Due to its many roles in tumorigenesis and metastatic
processes, the CCL2-CCR2 signaling axis is currently being
pursued as a potential therapeutic target for cancer.},
keywords = {Cell Line, Tumor / Chemokine CCL2: genetics / Chemokines /
Ligands / Receptors, CCR2: genetics / Tumor Microenvironment
/ Angiogenesis (Other) / CCL2 (Other) / CCR2 (Other) /
Cancer (Other) / Extravasation (Other) / Immunity (Other) /
Immunosuppression (Other) / Invasion (Other) / MCP-1 (Other)
/ Macrophage (Other) / Metastasis (Other) / Microenvironment
(Other) / NFκB (Other) / TAM (Other) / Tumor (Other) /
Chemokine CCL2 (NLM Chemicals) / Chemokines (NLM Chemicals)
/ Ligands (NLM Chemicals) / Receptors, CCR2 (NLM Chemicals)},
cin = {F180},
ddc = {570},
cid = {I:(DE-He78)F180-20160331},
pnm = {316 - Infektionen, Entzündung und Krebs (POF4-316)},
pid = {G:(DE-HGF)POF4-316},
typ = {PUB:(DE-HGF)3 / PUB:(DE-HGF)16},
pubmed = {pmid:34286437},
doi = {10.1007/978-3-030-62658-7_1},
url = {https://inrepo02.dkfz.de/record/169932},
}