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000170086 0247_ $$2doi$$a10.1038/s41416-021-01432-8
000170086 0247_ $$2pmid$$apmid:34341517
000170086 0247_ $$2ISSN$$a0007-0920
000170086 0247_ $$2ISSN$$a1532-1827
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000170086 037__ $$aDKFZ-2021-01749
000170086 041__ $$aEnglish
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000170086 1001_ $$00000-0001-8055-3729$$aPark, Hanla A$$b0$$eFirst author
000170086 245__ $$aMendelian randomisation study of smoking exposure in relation to breast cancer risk.
000170086 260__ $$aEdinburgh$$bNature Publ. Group$$c2021
000170086 3367_ $$2DRIVER$$aarticle
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000170086 500__ $$a#EA:C020#LA:C020# /2021 Oct;125(8):1135-1145
000170086 520__ $$aDespite a modest association between tobacco smoking and breast cancer risk reported by recent epidemiological studies, it is still equivocal whether smoking is causally related to breast cancer risk.We applied Mendelian randomisation (MR) to evaluate a potential causal effect of cigarette smoking on breast cancer risk. Both individual-level data as well as summary statistics for 164 single-nucleotide polymorphisms (SNPs) reported in genome-wide association studies of lifetime smoking index (LSI) or cigarette per day (CPD) were used to obtain MR effect estimates. Data from 108,420 invasive breast cancer cases and 87,681 controls were used for the LSI analysis and for the CPD analysis conducted among ever-smokers from 26,147 cancer cases and 26,072 controls. Sensitivity analyses were conducted to address pleiotropy.Genetically predicted LSI was associated with increased breast cancer risk (OR 1.18 per SD, 95% CI: 1.07-1.30, P = 0.11 × 10-2), but there was no evidence of association for genetically predicted CPD (OR 1.02, 95% CI: 0.78-1.19, P = 0.85). The sensitivity analyses yielded similar results and showed no strong evidence of pleiotropic effect.Our MR study provides supportive evidence for a potential causal association with breast cancer risk for lifetime smoking exposure but not cigarettes per day among smokers.
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