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@ARTICLE{Mori:177565,
author = {N. Mori and P. Keski-Rahkonen and A. Gicquiau and S.
Rinaldi and N. Dimou and S. Harlid and J. Harbs and B. Van
Guelpen and D. Aune and A. J. Cross and K. K. Tsilidis and
G. Severi and M. Kvaskoff and A. Fournier and R. Kaaks$^*$
and R. Turzanski-Fortner$^*$ and M. B. Schulze and P.
Jakszyn and M.-J. Sánchez and S. M. Colorado-Yohar and E.
Ardanaz and R. Travis and E. L. Watts and G. Masala and V.
Krogh and R. Tumino and C. Sacerdote and S. Panico and B.
Bueno-de-Mesquita and I. T. Gram and M. Waaseth and M. J.
Gunter and N. Murphy},
title = {{E}ndogenous {C}irculating {S}ex {H}ormone {C}oncentrations
and {C}olon {C}ancer {R}isk in {P}ostmenopausal {W}omen: {A}
{P}rospective {S}tudy and {M}eta-{A}nalysis.},
journal = {JNCI cancer spectrum},
volume = {5},
number = {6},
issn = {2515-5091},
address = {Oxford},
publisher = {Oxford University Press},
reportid = {DKFZ-2021-02632},
pages = {pkab084},
year = {2021},
abstract = {Observational studies have consistently reported that
postmenopausal hormone therapy use is associated with lower
colon cancer risk, but epidemiologic studies examining the
associations between circulating concentrations of
endogenous estrogens and colorectal cancer have reported
inconsistent results.We investigated the associations
between circulating concentrations of estrone, estradiol,
free estradiol, testosterone, free testosterone,
androstenedione, dehydroepiandrosterone (DHEA),
progesterone, and sex hormone-binding globulin (SHBG) with
colon cancer risk in a nested case-control study of 1028
postmenopausal European women (512 colon cancer cases, 516
matched controls) who were noncurrent users of exogenous
hormones at blood collection. Multivariable conditional
logistic regression models were used to compute odds ratios
and $95\%$ confidence intervals to evaluate the association
between circulating sex hormones and colon cancer risk. We
also conducted a dose-response meta-analysis of prospective
studies of circulating estrone and estradiol with
colorectal, colon, and rectal cancer risk in postmenopausal
women. All statistical tests were 2-sided.In the
multivariable model, a nonstatistically significantly
positive relationship was found between circulating estrone
and colon cancer risk (odds ratio per log2 1-unit increment
= 1.17 $[95\%$ confidence interval = 1.00 to 1.38]; odds
ratioquartile4-quartile1 = 1.33 $[95\%$ confidence interval
= 0.89 to 1.97], P trend = .20). Circulating concentrations
of estradiol, free estradiol, testosterone, free
testosterone, androstenedione, DHEA, progesterone, and SHBG
were not associated with colon cancer risk. In the
dose-response meta-analysis, no clear evidence of
associations were found between circulating estradiol and
estrone concentrations with colorectal, colon, and rectal
cancer risk.Our observational and meta-analysis results do
not support an association between circulating
concentrations of endogenous sex hormones and colon or
rectal cancer in postmenopausal women.},
cin = {C020},
ddc = {610},
cid = {I:(DE-He78)C020-20160331},
pnm = {313 - Krebsrisikofaktoren und Prävention (POF4-313)},
pid = {G:(DE-HGF)POF4-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:34805742},
pmc = {pmc:PMC8598284},
doi = {10.1093/jncics/pkab084},
url = {https://inrepo02.dkfz.de/record/177565},
}