TY - JOUR
AU - Hanna, Bola S
AU - Llaó-Cid, Laura
AU - Iskar, Murat
AU - Roessner, Philipp M
AU - Klett, Lara C
AU - Wong, John K L
AU - Paul, Yashna
AU - Ioannou, Nikolaos
AU - Öztürk, Selcen
AU - Mack, Norman
AU - Kalter, Verena
AU - Colomer, Dolors
AU - Campo, Elías
AU - Bloehdorn, Johannes
AU - Stilgenbauer, Stephan
AU - Dietrich, Sascha
AU - Schmidt, Manfred
AU - Gabriel, Richard
AU - Rippe, Karsten
AU - Feuerer, Markus
AU - Ramsay, Alan G
AU - Lichter, Peter
AU - Zapatka, Marc
AU - Seiffert, Martina
TI - Interleukin-10 receptor signaling promotes the maintenance of a PD-1int TCF-1+ CD8+ T cell population that sustains anti-tumor immunity.
JO - Immunity
VL - 54
IS - 12
SN - 1074-7613
CY - New York, NY
PB - Elsevier
M1 - DKFZ-2021-03072
SP - 2825-2841.e10
PY - 2021
N1 - #EA:B060#LA:B060# / 2021 Dec 14;54(12):2825-2841.e10
AB - T cell exhaustion limits anti-tumor immunity and responses to immunotherapy. Here, we explored the microenvironmental signals regulating T cell exhaustion using a model of chronic lymphocytic leukemia (CLL). Single-cell analyses identified a subset of PD-1hi, functionally impaired CD8+ T cells that accumulated in secondary lymphoid organs during disease progression and a functionally competent PD-1int subset. Frequencies of PD-1int TCF-1+ CD8+ T cells decreased upon Il10rb or Stat3 deletion, leading to accumulation of PD-1hi cells and accelerated tumor progression. Mechanistically, inhibition of IL-10R signaling altered chromatin accessibility and disrupted cooperativity between the transcription factors NFAT and AP-1, promoting a distinct NFAT-associated program. Low IL10 expression or loss of IL-10R-STAT3 signaling correlated with increased frequencies of exhausted CD8+ T cells and poor survival in CLL and in breast cancer patients. Thus, balance between PD-1hi, exhausted CD8+ T cells and functional PD-1int TCF-1+ CD8+ T cells is regulated by cell-intrinsic IL-10R signaling, with implications for immunotherapy.
KW - CD8(+) T cells (Other)
KW - CLL (Other)
KW - IL-10 (Other)
KW - IL-10R (Other)
KW - NFAT (Other)
KW - PD-1 heterogeneity (Other)
KW - STAT3 (Other)
KW - T cell exhaustion (Other)
KW - TCF-1 (Other)
KW - tumor microenvironment (Other)
LB - PUB:(DE-HGF)16
C6 - pmid:34879221
DO - DOI:10.1016/j.immuni.2021.11.004
UR - https://inrepo02.dkfz.de/record/178063
ER -