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@ARTICLE{Huan:179931,
author = {T. Huan and S. Nguyen and E. Colicino and C. Ochoa-Rosales
and W. D. Hill and J. A. Brody and M. Soerensen and Y. Zhang
and A. Baldassari and M. A. Elhadad and T. Toshiko and Y.
Zheng and A. Domingo-Relloso and D. H. Lee and J. Ma and C.
Yao and C. Liu and S.-J. Hwang and R. Joehanes and M.
Fornage and J. Bressler and J. B. J. van Meurs and B.
Debrabant and J. Mengel-From and J. Hjelmborg and K.
Christensen and P. Vokonas and J. Schwartz and S. A. Gahrib
and N. Sotoodehnia and C. M. Sitlani and S. Kunze and C.
Gieger and A. Peters and M. Waldenberger and I. J. Deary and
L. Ferrucci and Y. Qu and P. Greenland and D. M. Lloyd-Jones
and L. Hou and S. Bandinelli and T. Voortman and B. Hermann
and A. Baccarelli and E. Whitsel and J. S. Pankow and D.
Levy},
title = {{I}ntegrative analysis of clinical and epigenetic
biomarkers of mortality.},
journal = {Aging cell},
volume = {21},
number = {6},
issn = {1474-9718},
address = {Oxford [u.a.]},
publisher = {Wiley-Blackwell},
reportid = {DKFZ-2022-00980},
pages = {e13608},
year = {2022},
note = {2022 Jun;21(6):e13608},
abstract = {DNA methylation (DNAm) has been reported to be associated
with many diseases and with mortality. We hypothesized that
the integration of DNAm with clinical risk factors would
improve mortality prediction. We performed an epigenome-wide
association study of whole blood DNAm in relation to
mortality in 15 cohorts (n = 15,013). During a mean
follow-up of 10 years, there were 4314 deaths from all
causes including 1235 cardiovascular disease (CVD) deaths
and 868 cancer deaths. Ancestry-stratified meta-analysis of
all-cause mortality identified 163 CpGs in European ancestry
(EA) and 17 in African ancestry (AA) participants at p < 1
× 10-7 , of which 41 (EA) and 16 (AA) were also associated
with CVD death, and 15 (EA) and 9 (AA) with cancer death. We
built DNAm-based prediction models for all-cause mortality
that predicted mortality risk after adjusting for clinical
risk factors. The mortality prediction model trained by
integrating DNAm with clinical risk factors showed an
improvement in prediction of cancer death with $5\%$
increase in the C-index in a replication cohort, compared
with the model including clinical risk factors alone.
Mendelian randomization identified 15 putatively causal CpGs
in relation to longevity, CVD, or cancer risk. For example,
cg06885782 (in KCNQ4) was positively associated with risk
for prostate cancer (Beta = 1.2, PMR = 4.1 × 10-4 ) and
negatively associated with longevity (Beta = -1.9, PMR =
0.02). Pathway analysis revealed that genes associated with
mortality-related CpGs are enriched for immune- and
cancer-related pathways. We identified replicable DNAm
signatures of mortality and demonstrated the potential
utility of CpGs as informative biomarkers for prediction of
mortality risk.},
keywords = {DNA methylation (Other) / cancer (Other) / cardiovascular
disease (Other) / machine learning (Other) / mortality
(Other)},
cin = {C070},
ddc = {610},
cid = {I:(DE-He78)C070-20160331},
pnm = {313 - Krebsrisikofaktoren und Prävention (POF4-313)},
pid = {G:(DE-HGF)POF4-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:35546478},
doi = {10.1111/acel.13608},
url = {https://inrepo02.dkfz.de/record/179931},
}