TY - JOUR AU - Murad, Shafiq AU - Michen, Susanne AU - Becker, Alexander AU - Füssel, Monika AU - Schackert, Gabriele AU - Tonn, Torsten AU - Momburg, Frank AU - Temme, Achim TI - NKG2C+ NK Cells for Immunotherapy of Glioblastoma Multiforme. JO - International journal of molecular sciences VL - 23 IS - 10 SN - 1422-0067 CY - Basel PB - Molecular Diversity Preservation International M1 - DKFZ-2022-01119 SP - 5857 PY - 2022 AB - In glioblastoma, non-classical human leucocyte antigen E (HLA-E) and HLA-G are frequently overexpressed. HLA-E loaded with peptides derived from HLA class I and from HLA-G contributes to inhibition of natural killer (NK) cells with expression of the inhibitory receptor CD94/NKG2A. We investigated whether NK cells expressing the activating CD94/NKG2C receptor counterpart were able to exert anti-glioma effects. NKG2C+ subsets were preferentially expanded by a feeder cell line engineered to express an artificial disulfide-stabilized trimeric HLA-E ligand (HLA-E*spG). NK cells expanded by a feeder cell line, which facilitates outgrowth of conventional NKG2A+, and fresh NK cells, were included for comparison. Expansion via the HLA-E*spG feeder cells selectively increased the fraction of NKG2C+ NK cells, which displayed a higher frequency of KIR2DL2/L3/S2 and CD16 when compared to expanded NKG2A+ NK cells. NKG2C+ NK cells exhibited increased cytotoxicity against K562 and KIR:HLA-matched and -mismatched primary glioblastoma multiforme (GBM) cells when compared to NKG2A+ NK cells and corresponding fresh NK cells. Cytotoxic responses of NKG2C+ NK cells were even more pronounced when utilizing target cells engineered with HLA-E*spG. These findings support the notion that NKG2C+ NK cells have potential therapeutic value for treating gliomas. KW - HLA-E (Other) KW - HLA-G (Other) KW - NK cells (Other) KW - brain cancer (Other) KW - glioblastoma (Other) KW - immunotherapy (Other) LB - PUB:(DE-HGF)16 C6 - pmid:35628668 DO - DOI:10.3390/ijms23105857 UR - https://inrepo02.dkfz.de/record/180126 ER -