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@ARTICLE{Winkler:180331,
author = {T. W. Winkler and H. Rasheed and A. Teumer and M. Gorski
and B. X. Rowan and K. J. Stanzick and L. F. Thomas and A.
Tin and A. Hoppmann and A. Y. Chu and B. Tayo and C. H. L.
Thio and D. Cusi and J.-F. Chai and K. B. Sieber and K. Horn
and M. Li and M. Scholz and M. Cocca and M. Wuttke and P. J.
van der Most and Q. Yang and S. Ghasemi and T. Nutile and Y.
Li and G. Pontali and F. Günther and A. Dehghan and A.
Correa and A. Parsa and A. Feresin and A. P. J. de Vries and
A. B. Zonderman and A. V. Smith and A. J. Oldehinkel and A.
De Grandi and A. R. Rosenkranz and A. Franke and A. Teren
and A. Metspalu and A. A. Hicks and A. P. Morris and A.
Tönjes and A. Morgan and A. I. Podgornaia and A. Peters and
A. Körner and A. Mahajan and A. Campbell and B. I. Freedman
and B. Spedicati and B. Ponte and B. Schöttker$^*$ and B.
Brumpton and B. Banas and B. K. Krämer and B. Jung and B.
O. Åsvold and B. H. Smith and B. Ning and B. W. J. H.
Penninx and B. R. Vanderwerff and B. M. Psaty and C. M.
Kammerer and C. D. Langefeld and C. Hayward and C. N.
Spracklen and C. Robinson-Cohen and C. A. Hartman and C. M.
Lindgren and C. Wang and C. Sabanayagam and C.-K. Heng and
C. Lanzani and C.-C. Khor and C.-Y. Cheng and C. Fuchsberger
and C. Gieger and C. M. Shaffer and C.-A. Schulz and C. J.
Willer and D. I. Chasman and D. F. Gudbjartsson and D.
Ruggiero and D. Toniolo and D. Czamara and D. J. Porteous
and D. M. Waterworth and D. Mascalzoni and D. O.
Mook-Kanamori and D. F. Reilly and E. W. Daw and E. Hofer
and E. Boerwinkle and E. Salvi and E. P. Bottinger and E.-S.
Tai and E. Catamo and F. Rizzi and F. Guo$^*$ and F.
Rivadeneira and F. Guilianini and G. Sveinbjornsson and G.
Ehret and G. Waeber and G. Biino and G. Girotto and G.
Pistis and G. N. Nadkarni and G. E. Delgado and G. W.
Montgomery and H. Snieder and H. Campbell and H. D. White
and H. Gao and H. M. Stringham and H. Schmidt and H. Li and
H. Brenner$^*$ and H. Holm and H. Kirsten and H. Kramer and
I. Rudan and I. M. Nolte and I. Tzoulaki and I. Olafsson and
J. Martins and J. P. Cook and J. F. Wilson and J. Halbritter
and J. F. Felix and J. Divers and J. S. Kooner and J. J. Lee
and J. O'Connell and J. I. Rotter and J. Liu and J. Xu and
J. Thiery and J. Ärnlöv and J. Kuusisto and J.
Jakobsdottir and J. Tremblay and J. C. Chambers and J. B.
Whitfield and J. M. Gaziano and J. Marten and J. Coresh and
J. B. Jonas and J. C. Mychaleckyj and K. Christensen and
K.-U. Eckardt and K. L. Mohlke and K. Endlich and K.
Dittrich and K. A. Ryan and K. M. Rice and K. D. Taylor and
K. Ho and K. Nikus and K. Matsuda and K. Strauch and K.
Miliku and K. Hveem and L. Lind and L. Wallentin and L. M.
Yerges-Armstrong and L. M. Raffield and L. S. Phillips and
L. J. Launer and L.-P. Lyytikäinen and L. A. Lange and L.
Citterio and L. Klaric and M. A. Ikram and M. Ising and M.
E. Kleber and M. Francescatto and M. P. Concas and M. Ciullo
and M. Piratsu and M. Orho-Melander and M. Laakso and M.
Loeffler and M. Perola and M. H. de Borst and M. Gögele and
M. L. Bianca and M. A. Lukas and M. F. Feitosa and M. L.
Biggs and M. K. Wojczynski and M. Kavousi and M. Kanai and
M. Akiyama and M. Yasuda and M. Nauck and M. Waldenberger
and M.-L. Chee and M.-L. Chee and M. Boehnke and M. H.
Preuss and M. Stumvoll and M. A. Province and M. K. Evans
and M. L. O'Donoghue and M. Kubo and M. Kähönen and M.
Kastarinen and M. A. Nalls and M. Kuokkanen and M. Ghanbari
and M. Bochud and N. S. Josyula and N. G. Martin and N. Y.
Q. Tan and N. D. Palmer and N. Pirastu and N. Schupf and N.
Verweij and N. Hutri-Kähönen and N. Mononen and N. Bansal
and O. Devuyst and O. Melander and O. T. Raitakari and O.
Polasek and P. Manunta and P. Gasparini and P. P. Mishra and
P. Sulem and P. K. E. Magnusson and P. Elliott and P. M.
Ridker and P. Hamet and P. O. Svensson and P. K. Joshi and
P. Kovacs and P. P. Pramstaller and P. Rossing and P.
Vollenweider and P. van der Harst and R. Dorajoo and R. Z.
H. Sim and R. Burkhardt and R. Tao and R. Noordam and R.
Mägi and R. Schmidt and R. de Mutsert and R. Rueedi and R.
M. van Dam and R. J. Carroll and R. T. Gansevoort and R. J.
F. Loos and S. C. Felicita and S. Sedaghat and S.
Padmanabhan and S. Freitag-Wolf and S. A. Pendergrass and S.
E. Graham and S. D. Gordon and S.-J. Hwang and S. M. Kerr
and S. Vaccargiu and S. B. Patil and S. Hallan and S. J. L.
Bakker and S.-C. Lim and S. Lucae and S. Vogelezang and S.
Bergmann and T. Corre and T. S. Ahluwalia and T. Lehtimäki
and T. S. Boutin and T. Meitinger and T.-Y. Wong and T.
Bergler and T. J. Rabelink and T. Esko and T. Haller and U.
Thorsteinsdottir and U. Völker and V. H. X. Foo and V.
Salomaa and V. Vitart and V. Giedraitis and V. Gudnason and
V. W. V. Jaddoe and W. Huang and W. Zhang and W. B. Wei and
W. Kiess and W. März and W. Koenig and W. Lieb and X.
Gao$^*$ and X. Sim and Y. X. Wang and Y. Friedlander and
Y.-C. Tham and Y. Kamatani and Y. Okada and Y. Milaneschi
and Z. Yu and K. J. Stark and K. Stefansson and C. A. Böger
and A. M. Hung and F. Kronenberg and A. Köttgen and C.
Pattaro and I. M. Heid and C. H. L. Thio and P. J. van der
Most and M. H. de Borst and K. Ho and N. S. Josyula and S.
A. Pendergrass and B. X. Rowan and C. Robinson-Cohen and J.
M. Gaziano and L. S. Phillips and R. Tao and A. M. Hung},
collaboration = {L. c. study and D. study and V. M. V. Program},
title = {{D}ifferential and shared genetic effects on kidney
function between diabetic and non-diabetic individuals.},
journal = {Communications biology},
volume = {5},
number = {1},
issn = {2399-3642},
address = {London},
publisher = {Springer Nature},
reportid = {DKFZ-2022-01258},
pages = {580},
year = {2022},
abstract = {Reduced glomerular filtration rate (GFR) can progress to
kidney failure. Risk factors include genetics and diabetes
mellitus (DM), but little is known about their interaction.
We conducted genome-wide association meta-analyses for
estimated GFR based on serum creatinine (eGFR), separately
for individuals with or without DM (nDM = 178,691, nnoDM =
1,296,113). Our genome-wide searches identified (i) seven
eGFR loci with significant DM/noDM-difference, (ii) four
additional novel loci with suggestive difference and (iii)
28 further novel loci (including CUBN) by allowing for
potential difference. GWAS on eGFR among DM individuals
identified 2 known and 27 potentially responsible loci for
diabetic kidney disease. Gene prioritization highlighted 18
genes that may inform reno-protective drug development. We
highlight the existence of DM-only and noDM-only effects,
which can inform about the target group, if respective genes
are advanced as drug targets. Largely shared effects suggest
that most drug interventions to alter eGFR should be
effective in DM and noDM.},
cin = {C070},
ddc = {570},
cid = {I:(DE-He78)C070-20160331},
pnm = {313 - Krebsrisikofaktoren und Prävention (POF4-313)},
pid = {G:(DE-HGF)POF4-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:35697829},
doi = {10.1038/s42003-022-03448-z},
url = {https://inrepo02.dkfz.de/record/180331},
}