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@ARTICLE{Eskilsson:181427,
author = {E. Eskilsson and G. V. Røsland and G. M. Solecki$^*$ and
Q. Wang and P. N. Harter and G. Graziani and R. G. W.
Verhaak and F. Winkler$^*$ and R. Bjerkvig and H. Miletic},
title = {{EGFR} heterogeneity and implications for therapeutic
intervention in glioblastoma.},
journal = {Neuro-Oncology},
volume = {20},
number = {6},
issn = {1522-8517},
address = {Oxford},
publisher = {Oxford Univ. Press},
reportid = {DKFZ-2022-01995},
pages = {743 - 752},
year = {2018},
note = {POT Topic: 317},
abstract = {Patients with glioblastoma (GBM) have a universally poor
prognosis and are in urgent need of effective treatment
strategies. Recent advances in sequencing techniques
unraveled the complete genomic landscape of GBMs and
revealed profound heterogeneity of individual tumors even at
the single cell level. Genomic profiling has detected
epidermal growth factor receptor (EGFR) gene alterations in
more than half of GBMs. Major genetic events include
amplification and mutation of EGFR. Yet, treatment
strategies targeting EGFR have thus far failed in clinical
trials. In this review, we discuss the clonal and functional
heterogeneity of EGFRs in GBM development and critically
reassess the potential of EGFRs as therapeutic targets.},
subtyp = {Review Article},
keywords = {ErbB Receptors: antagonists $\&$ inhibitors / ErbB
Receptors: genetics / Glioblastoma: drug therapy /
Glioblastoma: genetics / Glioblastoma: pathology / Humans /
Molecular Targeted Therapy / Mutation / Prognosis / Protein
Kinase Inhibitors: therapeutic use / Protein Kinase
Inhibitors (NLM Chemicals) / EGFR protein, human (NLM
Chemicals) / ErbB Receptors (NLM Chemicals)},
cin = {G370},
ddc = {610},
cid = {I:(DE-He78)G370-20160331},
pnm = {317 - Translational cancer research (POF3-317)},
pid = {G:(DE-HGF)POF3-317},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:29040782},
pmc = {pmc:PMC5961011},
doi = {10.1093/neuonc/nox191},
url = {https://inrepo02.dkfz.de/record/181427},
}