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@ARTICLE{Ratiner:212429,
author = {K. Ratiner and T. Fachler-Sharp and E. Elinav$^*$},
title = {{S}mall {I}ntestinal {M}icrobiota {O}scillations, {H}ost
{E}ffects and {R}egulation-{A} {Z}oom into {T}hree {K}ey
{E}ffector {M}olecules.},
journal = {Biology},
volume = {12},
number = {1},
issn = {2079-7737},
address = {Basel},
publisher = {MDPI},
reportid = {DKFZ-2023-00148},
pages = {142},
year = {2023},
note = {#LA:F220#},
abstract = {The gut microbiota features a unique diurnal rhythmicity
which contributes to modulation of host physiology and
homeostasis. The composition and activity of the microbiota
and its secreted molecules influence the intestinal milieu
and neighboring organs, such as the liver. Multiple
immune-related molecules have been linked to the diurnal
microbiota-host interaction, including Reg3γ, IgA, and
MHCII, which are secreted or expressed on the gut surface
and directly interact with intestinal bacteria. These
molecules are also strongly influenced by dietary patterns,
such as high-fat diet and time-restricted feeding, which are
already known to modulate microbial rhythms and peripheral
clocks. Herein, we use Reg3γ, IgA, and MHCII as test cases
to highlight the divergent effects mediated by the diurnal
activity of the gut microbiota and their downstream host
effects. We further highlight current challenges and
conflicts, remaining questions, and perspectives toward a
holistic understanding of the microbiome's impacts on
circadian human behavior.},
subtyp = {Review Article},
keywords = {circadian clock (Other) / dietary timing (Other) /
microbiome (Other) / segmented filamentous bacteria (Other)
/ small intestine (Other)},
cin = {F220},
ddc = {570},
cid = {I:(DE-He78)F220-20160331},
pnm = {316 - Infektionen, Entzündung und Krebs (POF4-316)},
pid = {G:(DE-HGF)POF4-316},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:36671834},
pmc = {pmc:PMC9855434},
doi = {10.3390/biology12010142},
url = {https://inrepo02.dkfz.de/record/212429},
}