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@ARTICLE{Hamacher:274583,
author = {R. Hamacher$^*$ and H. Lanzafame$^*$ and I. A.
Mavroeidi$^*$ and K. M. Pabst$^*$ and L. Kessler$^*$ and P.
F. Cheung$^*$ and S. Bauer$^*$ and K. Herrmann$^*$ and H.-U.
Schildhaus$^*$ and J. Siveke$^*$ and W. P. Fendler$^*$},
title = {{F}ibroblast {A}ctivation {P}rotein {I}nhibitor
{T}heranostics: {T}he {C}ase for {U}se in {S}arcoma.},
journal = {PET clinics},
volume = {18},
number = {3},
issn = {1556-8598},
address = {Orlando, Fla. [u.a.]},
publisher = {Elsevier},
reportid = {DKFZ-2023-00660},
pages = {361-367},
year = {2023},
note = {2023 Jul;18(3):361-367},
abstract = {The theranostic use of fibroblast activation protein
inhibitors (FAPIs) is a novel approach in oncology. Sarcomas
are a heterogenous group of rare malignant tumors. Prognosis
remains poor in advanced/metastatic disease due to limited
therapeutic options. Sarcoma frequently demonstrate high
expression of fibroblast activation protein alpha on the
tumor cells themselves, in contrast to other solid tumors,
where it is mainly expressed on cancer-associated
fibroblasts. Consequently, high in vivo uptake of FAPI in
PET is observed in sarcoma. Moreover, retrospective case
reports and series demonstrated feasibility of FAPI
radioligand therapy with signs of tumor response.},
subtyp = {Review Article},
keywords = {FAP-Alpha (Other) / FAPI (Other) / Oncology (Other) / PET
(Other) / Radioligand (Other) / Sarcoma (Other) / Therapy
(Other)},
cin = {ED01},
ddc = {610},
cid = {I:(DE-He78)ED01-20160331},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:36997366},
doi = {10.1016/j.cpet.2023.02.008},
url = {https://inrepo02.dkfz.de/record/274583},
}