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@ARTICLE{Sher:276229,
author = {H. Sher and H. Sharif and T. Zaheer and S. A. Khan$^*$ and
A. Ali and H. Javed and A. Javed},
title = {{E}mploying computational tools to design a multi-epitope
vaccine targeting human immunodeficiency virus-1 ({HIV}-1).},
journal = {BMC genomics},
volume = {24},
number = {1},
issn = {1471-2164},
address = {Heidelberg},
publisher = {Springer},
reportid = {DKFZ-2023-01038},
pages = {276},
year = {2023},
abstract = {Despite being in the 21st century, the world has still not
been able to vanquish the global AIDS epidemic, and the only
foreseeable solution seems to be a safe and effective
vaccine. Unfortunately, vaccine trials so far have returned
unfruitful results, possibly due to their inability to
induce effective cellular, humoral and innate immune
responses. The current study aims to tackle these
limitations and propose the desired vaccine utilizing
immunoinformatic approaches that have returned promising
results in designing vaccines against various rapidly
mutating organisms. For this, all polyprotein and protein
sequences of HIV-1 were retrieved from the LANL (Los Alamos
National Laboratory) database. The consensus sequence was
generated after alignment and used to predict epitopes.
Conserved, antigenic, non-allergenic, T-cell inducing,
B-cell inducing, IFN-ɣ inducing, non-human homologous
epitopes were selected and combined to propose two vaccine
constructs i.e., HIV-1a (without adjuvant) and HIV-1b (with
adjuvant).HIV-1a and HIV-1b were subjected to antigenicity,
allergenicity, structural quality analysis, immune
simulations, and MD (molecular dynamics) simulations. Both
proposed multi-epitope vaccines were found to be antigenic,
non-allergenic, stable, and induce cellular, humoral, and
innate immune responses. TLR-3 docking and in-silico cloning
of both constructs were also performed.Our results indicate
HIV-1b to be more promising than HIV-1a; experimental
validations can confirm the efficacy and safety of both
constructs and in-vivo efficacy in animal models.},
keywords = {Acquired immunodeficiency syndrome (Other) / Bioinformatics
(Other) / Computational biology (Other) / Human
immunodeficiency virus (Other) / Immunity (Other) /
Immuno-informatics (Other) / Toll like receptor-3 (Other) /
Vaccinology (Other)},
cin = {HD01},
ddc = {610},
cid = {I:(DE-He78)HD01-20160331},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:37226084},
doi = {10.1186/s12864-023-09330-4},
url = {https://inrepo02.dkfz.de/record/276229},
}